Use the labels in the right column to find what you want. Or you can go thru them one by one, there are only 29,372 posts. Searching is done in the search box in upper left corner. I blog on anything to do with stroke. DO NOT DO ANYTHING SUGGESTED HERE AS I AM NOT MEDICALLY TRAINED, YOUR DOCTOR IS, LISTEN TO THEM. BUT I BET THEY DON'T KNOW HOW TO GET YOU 100% RECOVERED. I DON'T EITHER BUT HAVE PLENTY OF QUESTIONS FOR YOUR DOCTOR TO ANSWER.
Amen, I have done research on this matter. and there are alternatives that can be used in nearly endless combinations. i have had written a few break through protocols on a similar but more emergent issue: global ischemic damage from a post anoxic event. The major etiology of my concern is cytotoxicity and oxidative damage that occurs during tissue reperfusion. In in a case of strokes of the transient nature the damage will likely culmilative dependind on the amount of time after re-oxygination and the amount of tissue that has been deprived of O2. Also the rate of reperfusion plays a role in rehabilition and eventual prognosis, that is if the attending has any idea of what is actually going on. You are on the right track with the insight of D-cycloserine which can be an adjunct to other drugs such as amantadine and other NMDA antagonists and modulators. All Cannot be place in a post due to not being published at this point, but look closer at glycine channel agonist and their role with other agents.Sorry no time to proof read the message should be rewritten later in light of this.
Amen, I have done research on this matter. and there are alternatives that can be used in nearly endless combinations. i have had written a few break through protocols on a similar but more emergent issue: global ischemic damage from a post anoxic event. The major etiology of my concern is cytotoxicity and oxidative damage that occurs during tissue reperfusion. In in a case of strokes of the transient nature the damage will likely culmilative dependind on the amount of time after re-oxygination and the amount of tissue that has been deprived of O2. Also the rate of reperfusion plays a role in rehabilition and eventual prognosis, that is if the attending has any idea of what is actually going on. You are on the right track with the insight of D-cycloserine which can be an adjunct to other drugs such as amantadine and other NMDA antagonists and modulators. All Cannot be place in a post due to not being published at this point, but look closer at glycine channel agonist and their role with other agents.Sorry no time to proof read the message should be rewritten later in light of this.
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