Mst3b, a purine-sensitive Ste20-like protein kinase, regulates axon outgrowth

I wonder how this could be useful for neurogenesis, maybe to get white matter to connect to parts of the cortex. From 2006 so if we had any decent strategy/vision of stroke rehab the followup research could have provided positive or negative answers to questions of its worth.
http://www.pnas.org/content/103/48/18320.abstract

Abstract

The growth of axons is fundamental to the development and repair of brain circuitry. We show here that Mst3b, a neuron-specific homolog of the yeast kinase Ste20, is critical for axon outgrowth. Mst3b is activated in response to trophic factors, and suppressing its expression (via siRNAs) or its function (by a dominant-negative mutant) blocks axon outgrowth. Inosine, a purine nucleoside that stimulates axon outgrowth, activates Mst3b kinase activity, whereas 6-thioguanine, a purine analog that blocks outgrowth, inhibits the activity of this kinase. These findings place Mst3b as a key regulator of axon outgrowth and help explain the purine sensitivity of this process.