If we had objective analysis of gait problems we could solve each individual problem. I know that's wishing for the moon but unless we demand results we aren't getting anywhere. So be a pain in the ass to your clinicians, they should know how to get you recovered.
http://www.sciencecodex.com/clinicians_should_pay_attention_to_stroke_patients_who_cannot_walk_at_36_mon_after_onset-115583
Gait dysfunction is one of the most serious disabling sequelae of
stroke. Regaining gait ability in stroke is a primary goal of
neurorehabilitation. Furthermore, gait is a less demanding motor
function than hand function. Stroke patients can walk when motor
function is recovered in the proximal joint (hip and knee), at least to
the degree of being able to oppose gravity. In general, most motor
recovery after stroke occurs within 3-6 months after onset, and gait
function usually recovers within 3 months of stroke onset. Therefore,
clinicians need to look for the cause of gait inability and perform
intensive rehabilitation for stroke patients who cannot walk after 3-6
months after insult. Sung Ho Jang and team from the College of Medicine,
Yeungnam University (Daegu, Republic of Korea) reported on a stroke
patient who showed delayed gait recovery between 8 and 11 months after
the onset of intracerebral hemorrhage, which has been reported in the Neural Regeneration Research
(Vol. 8, No. 16, 2013).
This 32-year-old female patient underwent
craniotomy and drainage for right intracerebral hemorrhage due to
rupture of an arteriovenous malformation. Brain MRI revealed a large
leukomalactic lesion in the right fronto-parietal cortex. Diffusion
tensor tractography at 8 months after onset revealed that the right
corticospinal tract was severely injured. At this time, the patient
could not stand or walk despite undergoing rehabilitation from 2 months
after onset. It was believed that severe spasticity of the left leg and
right ankle was largely responsible, and thus, antispastic drugs,
antispastic procedures (alcohol neurolysis of the motor branch of the
tibial nerve and an intramuscular alcohol wash of both tibialis
posterior muscles) and physical therapy were tried to control the
spasticity.
These measures relieved the severe spasticity, with the
result that the patient was able to stand at 3 months. In addition, the
improvements in sensorimotor function, visuospatial function, and
cognition also seemed to contribute to gait recovery. As a result, she
gained the ability to walk independently on even floor with a left ankle
foot orthosis at 11 months after onset. This case illustrates that
clinicians should attempt to find the cause of gait inability and to
initiate intensive rehabilitation in stroke patients who cannot walk at
3-6 months after onset.
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