C-reactive protein and cognition are unrelated to leukoaraiosis

My doctor just threw off a comment to me that I had had some earlier infarcts, so I'm assuming it was some white matter hyperintensities, obviously not important since I didn't get to see a picture of them or any explanation of what to do about them. My doctor told me nothing and as far as I could tell knew nothing, did nothing and should have never been paid for his non-efforts. So even though I have leukoaraiosis I think my cognition is still damn good, better than my doctors.
http://scholar.google.com/scholar_url?hl=en&q=http://downloads.hindawi.com/journals/tswj/aip/121679.pdf&sa=X&scisig=AAGBfm1sH0XbW4TGUMjDn5PEQgJ5eR3ILA&oi=scholaralrt
Introduction
Inflammation has been increasingly recognized as component in cerebrovascular (1) and neurodegenerative diseases (2, 3). In addition, biological aging of the brain is partly attributable to aging of the cerebrovascular circulation and the effects of vascular changes on the brain (4). Inflammation has been linked to the pathogenesis of cardiovascular disease, obesity and insulin resistance, which are so related to cognitive impairment (5). The hypothesis that inflammation is related to cognitive impairment, although new, is consistent (2). Therefore, few studies evaluated that circulating inflammatory proteins are associated with increased risk of dementia (6), cognitive impairment (7) and cerebral white matter lesions (WML), common referred to leukoaraiosis (8-10).
CRP, composed of five 23 kDa subunits, is a hepatically derived pentraxin that has important role in the human immune system (11). That protein is a sensitive nonspecific marker of systemic low-grade inflammation (5) and increased serum concentrations of CRP have been associated with impaired cognition, stroke and depression (2, 6, 8).

Beyond pro-inflammatory response, that causes neuronal damage directly, increased concentrations of CRP acting as cardiovascular risk factor - approved predictor by Food and Drug Administration - or causing brain atherosclerosis can result in cerebral macro or microangiopathies. Both lesions can disrupt the integrity of frontal-subcortical circuits and are responsible for the development of cognitive impairment, dementia or depressive disorders (12). There are some evidences that elevated serum CRP levels may be a useful biomarker to identify individuals at an increased risk for cognitive impairment (7).