Is stroke rehabilitation a metabolic problem?

Don't just lazily post a question, suggest a solution or next steps. Do you expect survivors to solve this on their own?
http://search.naric.com/research/rehab/redesign_record.cfm?search=2&type=all&criteria=J68283&phrase=no&rec=124236
NARIC Accession Number: J68283.  What's this?
ISSN: 0269-9052.
Author(s): Aquilani, Roberto; Boselli, Mirella; Paola, Baiardi; Pasini, Evasio; Iadarola, Paola; Verri, Manuela; Viglio, Simona; Condino, Annamaria; Boschi, Federica.
Publication Year: 2014.
Number of Pages: 13.
Abstract: Study investigated the impact of inflammation during the rehabilitation stage of strokes and its effect on neurofunctional recovery. Anthropometric characteristics, serum proteins and inflammatory markers, plasma amino acids, and neurofunction were all assessed in 94 patients suffering from strokes and admitted to rehabilitation. An inflammatory status was found in 52 (55.3 percent) of the patients. Inflammation was positively linked to positive proteins (alpha-1 globulin) and negatively linked to negative proteins (albumin, prealbumin, transferrin) of the acute-phase response. Inflammation was associated with low plasma concentrations of total amino acids. For the multiple logistic regression analysis, albumin and body weight maintenance were independent predictors of patient functional independence. Inflammation in dysphagic stroke patients was associated with more accentuated disability compared to non-inflamed dysphagics. The serum positive reactant alpha-1 globulin was the most powerful predictor of dysphagia severity. At discharge, dysphagia improvement was associated with improved acute-phase negative proteins. The results indicate that an inflammatory status may persist for most patients with strokes during the rehabiliation stage of the disease and its prevalence is higher in dysphagic compared to non-dysphagic subjects.