How Stress Promotes Atherosclerosis

You will need to demand that your doctor remove all the stressors from your hospital environment.
1. Poor sleep
2. No objective diagnosis of damage.
3. No path to 100% recovery.
4. Nocebo comments about no further recovery/plateau.
5. Worries about cognitive decline with no stroke protocol to fix that.
6. Fatigue.
7. Spasticity which your doctor has no idea how to cure.

http://www.united-academics.org/magazine/health-medicine/how-stress-promotes-atherosclerosis/
Atherosclerosis is an inflammatory disease characterized by the “hardening” of the arteries. This is caused by the accumulation of deposits mainly composed of cell debris, fats, cholesterol, calcium, on the inner walls of arteries leading to vessels’ narrowing and decreased blood flow. These deposits are called plaques and they also contain platelets and immune cells (leukocytes), in particular inflammatory monocytes and macrophages. Enzymes produced by the immune cells can partially degrade the plaques causing their disruption and cause formation of blood clots that will occlude the vessels and block oxygen supply to different organs and tissues. If a clot occurs in vessels of the heart or the brain, it will elicit a heart attack or a stroke respectively.
Link between stress and atherosclerosis
There is evidence that chronic stress increases the risk of atherosclerosis, but no mechanism linking the two phenomena has been demonstrated so far. Since stressful emotional states can affect the function of the immune system, Heidt and colleagues of the Massachusetts General Hospital hypothesized that stress increases the activity of inflammatory cells in the plaques facilitating their rupture, as you can read in their recently published article.
To test this possibility the scientists measured the effects of various forms of stress (including isolation, damp bedding, overnight illumination) on the number of leukocytes in mice. They found that compared to the controls the stressed animals had significantly more circulating immune cells. This was the result of increased division of the hematopoietic stem cells (HSCs), which are the immature precursors of the immune cells, and subsequent increased production of leukocyte progenitors in their bone marrow.

More at link.