Saturday, July 20, 2019

Nutraceuticals and physical activity: their role on oxysterols-mediated neurodegeneration

Your doctor is responsible for deciphering this and creating a  protocol to prevent dementia and Parkinsons.

You will need this. SO DEMAND TO KNOW FROM YOUR DOCTOR AND STROKE HOSPITAL WHAT THEY ARE DOING TO ENSURE THIS CREATES A PREVENTION PROTOCOL FOR YOUR USE. 


Your chances of getting dementia.

1. A documented 33% dementia chance post-stroke from an Australian study?   May 2012.

2. Then this study came out and seems to have a range from 17-66%. December 2013.

3. A 20% chance in this research.   July 2013.

4. Dementia Risk Doubled in Patients Following Stroke September 2018 

5. Parkinson’s Disease May Have Link to Stroke March 2017

 

The latest here:


Nutraceuticals and physical activity: their role on oxysterols-mediated neurodegeneration

Highlights

The role of nutraceuticals and physical activity on oxysterol-mediated neurodegeneration was reviewed.
Nutraceutical effects have been reported in the counteraction of oxysterol effects at CNS cells level.
Physical activity might become potential supporting strategy in the counteraction of oxysterol effects at brain level.

Abstract

Over the past few years, the contribution of oxysterols to the onset and development of some of the major neurodegenerative diseases (such as Alzheimer’s and Parkinson’s diseases) has been scientifically asserted, being mainly related to altered brain cholesterol homeostasis. To counteract oxysterol induced inflammation at neuronal level, one possible intervention approach is the administration of some nutrients and/or plant secondary metabolites. On the other hand, the pleiotropic beneficial effects of physical activity seem to play an important role on prevention and counteraction of neurodegenerative diseases, through the modulation of oxysterol homeostasis and the prevention of demyelination. The present review provides a picture of the promising role of nutraceuticals and physical activity on oxysterol-mediated neurodegeneration, pointing out also the different in vitro and in vivo aspects that need to be further investigated for a better understanding of the association of these three counterparts and their overall effect on people at increased risk for neurodegenerative diseases.

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