Maybe you want to ask your doctor about these possible solutions to stiff arteries. Why would you listen to me? I'm a non-medical person.
You can test if you have stiff arteries here and here.
Watermelon juice reverses hardening of the arteries
Stiff arteries relax like younger blood vessels after taking alagebrium
http://www.alphagalileo.org/ViewItem.aspx?ItemId=142222&CultureCode=en
High blood pressure is highly age-related and affects more than 1
billion people worldwide. But doctors can’t fully explain the cause of
90 per cent of all cases. A computer model of a “virtual human”
suggests that stiff arteries alone are enough to cause high blood
pressure.
“Our results suggest that arterial stiffness represents a major
therapeutic target. This is contrary to existing models, which typically
explain high blood pressure in terms of defective kidney function,”
says Klas Pettersen, a researcher at the Norwegian University of Life
Sciences and first author of the study, recently published in PLOS
Computational Biology.
High blood pressure is a major source of morbidity and mortality,
because it makes individuals more prone to heart failure, stroke and
kidney disease.
When blood pressure travels down the aorta from the heart, a special
group of cells in the aortic wall, called baroreceptors, sense the
pressure in this stretch of the aortic wall and send signals with this
information to the nervous system.
If the blood pressure is too high, these cells send stronger signals
and the body is able to lower blood pressure. However, if the aorta gets
stiffer, as typically happens with age, this stretch of the aorta is
not as sensitive as it once was in measuring blood pressure. Thus,
although a person’s blood pressure may have increased, the baroreceptors
do not signal as intensively as they should and the body does not get
the message to lower blood pressure.
“With the stiffening of the wall that follows ageing, these sensors
become less able to send signals that reflect the actual blood pressure.
Our mathematical model predicts the quantitative effects of this
process on blood pressure,” says Pettersen.
“If our hypothesis is proven right, arterial stiffness and
baroreceptor signaling will become hotspot targets for the treatment of
high blood pressure and the development of new medicines and medical
devices,” says Stig W. Omholt from the Norwegian University of Science
and Technology, who was the senior investigator of the research project.
With the use of existing experimental data and models of the aging
human aorta, the researchers were able to show quantitatively how the
stiffening of the aorta with age causes the baroreceptors to misinform
the central nervous system about blood pressure, thus preventing the
system from downregulating blood pressure. The model predictions were
compared with data from the Nord-Trøndelag Health Study (HUNT2), which
is comprised of information on the health history of 74,000 people,
including blood sample collection from 65,000 people.
NTNU’s Omholt says that the study is a good example of how very
complex human disease can be understood by use of mathematical models
and thus allow for much better treatment strategies.
“If we are to succeed in developing predictive, preventive and
participatory medicine envisioned by so many, there is no substitute
for building much stronger transdisciplinary ties between the life
sciences, the mathematical sciences and engineering across the whole
spectrum of basic, translational and applied research. And mathematical
models of the human physiology will be at the core of this
development,” he says.
In September the Norwegian University of Science and Technology will
host the biannual conference on the Virtual Physiological Human, where
this research will be presented. http://www.ntnu.edu/vph2014
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