Coconut Oil Supplementation: an innovative strategy for cardiovascular disease risk reduction

I'm sure there will be NO followup to determine what a CVD risk reduction diet should look like. Everything is too fragmented for laypersons to figure this out. And your stroke medical 'professionals' who are paid to know this stuff won't do it because they are

WAITING FOR SOMEONE ELSE TO SOLVE THE PROBLEM?  Dammed lazy assholes.

 

Stop waiting and start doing, your stroke patients are expecting you to help them. 

Coconut Oil Supplementation: an innovative strategy for cardiovascular disease risk reduction

1. Claudia Thompson-Felty,
2. Carol S Johnston and
3. Ashley Ryder

+ Author Affiliations

1. Arizona State University, Phoenix, AZ

Abstract

The American Heart Association has projected that by 2030, 40.5% of the US population will have some form of cardiovascular disease (CVD). Elevated blood cholesterol, high blood pressure, and smoking are major risk factors for CVD. The current dietary recommendations to improve HDL cholesterol include drinking alcohol in moderation and choosing healthier dietary fats (monounsaturated and polyunsaturated) while limiting saturated fat to less than 7% total daily energy. Conversely, some research suggests that consuming 20–30% of energy as coconut oil (CO), which is high in saturated fat, may raise HDL. In secondary data analyses of a fish oil (FO) supplementation trial, we noted a significant improvement in HDL in healthy adults supplementing only 2 g CO daily for 8 weeks (~1% total daily energy). Forty-eight healthy individuals (24 FO and 24 CO, 12/36 M/F; 26.5±8.5 y, BMI, 24.5±3.6 kg/m²) were stratified by blood type, gender, age, and BMI and randomly assigned to one of two supplement groups: FO, 2000 mg/d, or CO, 2000 mg/d. At baseline, there were no significant mean differences in total cholesterol, LDL, HDL, or triglycerides between the groups. CO supplementation improved HDL cholesterol in comparison to FO (+4.5±7.9 vs. −0.6±6.9 mg/dL, p=0.026). Changes in LDL cholesterol and the total cholesterol/HDL ratio did not differ between groups (+3.6±19.0 vs. +1.9±18.7 mg/dL and −0.13±0.48 vs. +0.01±0.31 mg/dL respectively). These data suggest that much smaller amounts of CO than previously reported can significantly raise HDL without adversely impacting LDL or total cholesterol; however, more research is needed to confirm these secondary findings.

Support or Funding Information

Graduate and Professional Student Association

Footnotes

• This abstract is from the Experimental Biology 2016 Meeting. There is no full text article associated with this abstract published in The FASEB Journal.