Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Sunday, October 26, 2025

Neuroplasticity: What Is It?

 But your doctor and hospital KNOW NOTHING ON HOW TO MAKE IT REPEATABLE ON DEMAND!

We don't SPECIFICALLY know why a neuron gives up its' current job and takes on a neighbors'.  Thus, nothing on neuroplasticity is scientifically repeatable on demand. So, DEMAND your doctor give you EXACT PROTOCOLS to use. Don't allow your doctor to give you generalities or guidelines like this article. 

Without that knowledge, telling us neuroplasticity will get us recovered IS A COMPLETE LOAD OF BULLSHIT!

Neuroplasticity: What Is It?


Chapter First Online: pp 197–207 Cite this chapter

Abstract

Neuroplasticity, the nervous system’s ability to undergo structural and functional changes, is fundamental to the brain’s adaptability. William James and Santiago Ramon y Cajal were early proponents of this concept, highlighting its role in habit formation and personality development. Research has since revealed complex mechanisms underlying neuroplasticity at molecular, cellular, and network levels. Long-term potentiation (LTP) and long-term depression (LTD) exemplify how synaptic efficiency is enhanced or diminished, demonstrating the bidirectional nature of synaptic plasticity.

Neuronal activity triggers structural modifications, such as spine formation and synaptic strengthening, influenced by recent and past experiences. Representational changes in cortical maps, as seen in somatosensory and motor cortex adaptations following sensory deprivation or amputation, further illustrate neuroplasticity. Cross-modal plasticity highlights the brain’s ability to repurpose regions for alternative functions, showcasing its flexibility.

Following brain injury, functional recovery is driven by regeneration of damaged tissue and reorganization of neural connections. While the central nervous system faces challenges due to inhibitory factors and scar formation, studies on neurogenesis in the hippocampus and olfactory bulb offer therapeutic insights. Reorganization involves surviving pathways compensating for lost functions, emphasizing the brain’s adaptability. Early intervention post-injury is crucial for guiding plastic changes toward recovery.

Understanding neuroplasticity mechanisms has significant implications for neurorehabilitation and regenerative medicine, potentially leading to effective interventions for brain injuries and neurodegenerative disorders, ultimately improving patient outcomes and quality of life.

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