Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Thursday, October 9, 2025

Dietary cholesterol intake and egg consumption in relation to all-cause and cardiovascular mortality after stroke

 But hasn't dietary cholesterol been proven not to be a CVD risk? Hasn't this horse been beaten to death multiple times?

Why Dietary Cholesterol Does Not Matter (For Most People) 

September 2023

Dietary cholesterol intake and egg consumption in relation to all-cause and cardiovascular mortality after stroke


Abstract

The findings on the associations of dietary cholesterol intake and egg consumption with mortality in the general population remain inconclusive. Our objective was to evaluate all-cause and cardiovascular mortality in relation to dietary cholesterol intake and egg consumption in a cohort of stroke survivors, a particularly vulnerable population. We conducted a prospective cohort analysis of 1367 stroke survivors recruited across the United States between 1999 and 2018. Dietary information was evaluated by a 24-h dietary recall at baseline obtained from NHANES while mortality data were derived from the National Death Index with follow-up through 2019. To investigate the associations of dietary cholesterol intake or egg consumption with all-cause and cardiovascular mortality, Cox proportional hazards and restricted cubic splines models were utilized. After a median follow-up of 79.0 months, the incidence per 1000 person-years was 59.9 (95% confidence interval [CI], 55.2–64.9) for all-cause and 23.3 (95% CI, 20.4–26.5) for cardiovascular mortality. Each 100 mg/1000 kcal/day elevation in cholesterol intake was related to raised risk of all-cause (hazard ratio [HR], 1.16; 95% CI, 1.05–1.27) and cardiovascular mortality (HR, 1.15; 95% CI, 1.00–1.31). A greater risk of all-cause mortality was shown in participants consuming > 1 egg/day (HR, 1.40; 95% CI, 1.06–1.84). The dose-response analysis highlighted the lowest risk of all-cause mortality when egg consumption was around 33.3 g/day. The observed correlations of dietary cholesterol intake or egg consumption with all-cause and cardiovascular mortality were stronger in the subgroups with cardiometabolic diseases (obesity, hypertension, diabetes mellitus, hyperlipidemia) or risk factors for cardiometabolic diseases (advanced age). Among stoke survivors, greater dietary cholesterol connects to an escalated risk of all-cause and cardiovascular mortality in a linear manner. Moderate egg consumption (≤ 1 egg/day) is relatively safe and excessive consumption links to an elevated risk of all-cause mortality.

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