Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Tuesday, October 14, 2025

Investigating cannabidiol's role in combating Alzheimer's-related neuroinflammation

Of course, your competent? doctor already has EXACT PROTOCOLS that address the neuroinflammation post stroke!

With your extra risk of dementia post stroke; DOES YOUR DOCTOR HAVE EXACT DEMENTIA PREVENTION PROTOCOLS? NO? So, your doctor is incompetent? 

1. A documented 33% dementia chance post-stroke from an Australian study?   May 2012.

2. Then this study came out and seems to have a range from 17-66%. December 2013.`    

3. A 20% chance in this research.   July 2013.

4. Dementia Risk Doubled in Patients Following Stroke September 2018 

  • neuroinflammation (53 posts to December 2015)
  • Do you prefer your doctor, hospital and board of director's incompetence NOT KNOWING? OR NOT DOING?

     Investigating cannabidiol's role in combating Alzheimer's-related neuroinflammation

    Neuroinflammation damages neurons and can contribute to diseases like Alzheimer's. Cannabidiol (CBD) has anti-inflammatory properties, which suggests that it could combat neuroinflammation in Alzheimer's. In a new eNeuro paper, Babak Baban and colleagues, from Augusta University, explored whether CBD can be leveraged as an antiinflammatory treatment in an established Alzheimer's disease mouse model. 

    The researchers assessed two distinct mechanisms for shaping immune responses and regulating neuroinflammation in the central nervous system following CBD treatment via inhalation. With several molecular and genetic measures, they discovered that CBD reduced expression of key regulators for neuroinflammation in Alzheimer's mice, which was associated with less proinflammatory molecules. Baban et al. also identified distinct regulators of the immune response and neuroinflammation with which CBD interacted. 

    Alzheimer's work has long centered on plaques and tangles. But our study shows that chronic autoinflammation is also a core driver of the disease. What's exciting is that CBD not only calms this immune overactivation but, in earlier work, we've shown it can also help clear plaques and tangles through a different mechanism. Together, this points to a multitarget approach with real therapeutic potential." 

    Babak Baban, Augusta University

    Source:
    Journal reference:

    Naeini, S. E., et al. (2025) Rethinking Alzheimer's: Harnessing Cannabidiol to Modulate IDO and cGAS Pathways for Neuroinflammation Control. eNeurodoi.org/10.1523/ENEURO.0114-25.2025


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