Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Monday, June 3, 2024

Brain Tissue Oxygenation

How EXACTLY is your doctor ensuring a good supply of oxygen to your brain immediately post stroke? Doing ANYTHING AT ALL?  

  • oxygen delivery (27 posts to January 2020) Many ideas in here, if your doctor isn't already using them to save neurons immediately post stroke; you don't have a functioning stroke doctor!

Brain Tissue Oxygenation

  • Chapter
  • First Online:
Principles and Practice of Neurocritical Care

Abstract

The brain represents 2% of body weight, but consumes 20% of the body’s oxygen supply as a result of its high metabolic demand. This chapter reviews the physiological determinants of cerebral oxygenation, and the role that ischaemia and hypoxia play in the two pathologies most commonly encountered in the neurocritical care unit - traumatic brain injury (TBI) and sub-arachnoid haemorrhage (SAH). It focuses primarily on the emerging technique of brain tissue oxygen monitoring (PbtO2) which is increasingly used to guide treatment in neurocritical care settings. Despite enormous potential, this method faces significant challenges associated with the complex interplay of systemic and local factors affecting cerebral perfusion and oxygenation. The chapter documents the evolution of PbtO2 monitoring and the evidence for its use. It illustrates how this technique has the potential to enhance clinical management strategies and significantly improve patient outcomes from acute brain injury.

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