Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Saturday, October 8, 2011

Zinc's role in the brain: Research gives insight into 50-year-old mystery

Anything to understand how the brain works.
http://medicalxpress.com/news/2011-10-zinc-role-brain-insight-year-old.html

Zinc plays a critical role in regulating how neurons communicate with one another, and could affect how memories form and how we learn. The new research, in the current issue of Neuron, was authored by Xiao-an Zhang, now a chemistry professor at the University of Toronto Scarborough (UTSC), and colleagues at MIT and Duke University.

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Researchers have been trying to pin down the role of zinc in the brain for more than fifty years, ever since scientists found high concentrations of the chemical in synaptic vesicles, a portion of the neuron that stores neurotransmitters. But it was hard to determine just what zinc's function was.

In the new work, the researchers designed a chemical called ZX1 that would bind with zinc rapidly after it was released from the vesicles but before it could complete its journey across the synapse. Using the chemical, they were able to observe how neurons behaved when deprived of zinc.

"As a chemist, I'm proud that I can make a contribution to neuroscience," says Zhang, who helped design the chemical while conducting postdoctoral research in Stephen J. Lippard's lab at MIT. He was joint first author of the paper, along with Enhui Pan from James O. McNamara's group at Duke University.

The researchers studied neurons in a brain region called the , which is associated with learning and . They found that removing zinc interfered with a process called long-term potentiation . Long-term potentiation strengthens the connection between two neurons, and seems to be important for memory and learning.

Zhang is currently working on developing new that could be used in medical imaging.

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