Showing posts with label doctor responsibility. Show all posts
Showing posts with label doctor responsibility. Show all posts
Wednesday, August 5, 2026
Tuesday, July 21, 2026
How Much Water Should You Drink Daily? Science-Backed Hydration Guide by Super Age
Another protocol for your doctor to have in place to make sure you leave the hospital better than you came in.
Which means you probably need to be catheterized or diapered immediately in the hospital. There aren't enough nurses to get all their patients safely to the bathroom in time. When I'm traveling, especially on airplanes and buses, I purposely stay dehydrated in order not to need to use the bathroom.
On coffee:
The caffeine in coffee gives it diuretic effects, meaning it causes your body to pass more urine. But these effects are too mild to cause dehydration, especially if you're a regular coffee drinker. Coffee may even be hydrating for some people, because it contains a lot of water.
On alcohol:
For reference, a standard drink—12 ounces of beer, 5 ounces of wine, 1.5 ounces of liquor—has 14 grams of alcohol, according to the National Institute on Alcohol Abuse and Alcoholism [3]. In other words, the alcohol alone in one standard drink can make your body produce a little less than half a cup of pee.
So to calculate; a standard 5 ounce glass of wine produces less than 4 ounces of pee. That doesn't sound like dehydration. Beer would make even better sense.
The latest here:
How Much Water Should You Drink Daily? Science-Backed Hydration Guide
Wednesday, July 8, 2026
Smart medical wristband tracks hidden arrhythmias to prevent strokes
This finds the problem, but does nothing to prevent strokes! What EXACTLY needs to occur to prevent that stroke? Bad title! Your doctor better EXACTLY KNOW THE ANSWER!
Smart medical wristband tracks hidden arrhythmias to prevent strokes
According to the World Health Organization, cardiovascular diseases claim almost 20 million lives every year and remain the leading cause of death worldwide. Although they are most often associated with heart attacks or strokes, serious complications do not always begin suddenly - sometimes their onset is far less noticeable.
For doctors, this raises a difficult question: how can they record something that is not happening during an examination? A standard electrocardiogram (ECG) shows the heart's activity only at a specific moment, while even longer-term monitoring does not always coincide with less frequent episodes of heart rhythm disorders. As a result, some arrhythmias, particularly short episodes of atrial fibrillation, may go unnoticed until they cause more serious health problems.
As society ages and the prevalence of chronic diseases rises, this diagnostic gap is becoming more significant. Patients need monitoring not only in healthcare settings but also in their daily lives. For physicians, it is important to understand not just whether an abnormal heart rhythm occurred during a specific examination, but also how frequently such episodes happen, how long they last, under what conditions they arise, and whether their occurrence increases over time.
A system developed by Kaunas University of Technology (KTU) and other Lithuanian researchers aims to address this problem, and its solutions are being applied in Teltonika Telemedic's TeltoHeart medical wristband. The system enables continuous heart rhythm monitoring, allows a more detailed ECG recording to be made using the same device and transmits the data to a doctor remotely.
Particularly important for post-stroke patients
Professor Dr Vaidotas Marozas, Director of the Biomedical Engineering Institute at KTU, emphasizes that the system was first developed with patients in mind for whom undetected heart rhythm disorders can have particularly serious consequences. One such group is people who have suffered a stroke.
The team chose to focus on post-stroke patients because atrial fibrillation is often short-lived and asymptomatic. As a result, standard examination methods - an electrocardiogram in a clinic or even Holter monitoring, where a patient wears a heart rhythm recording device for one or several days - often fail to detect such episodes."
Professor Dr Vaidotas Marozas, Director of the Biomedical Engineering Institute at KTU
According to the KTU professor, heart rhythm disorders, particularly atrial fibrillation, are directly linked to an increased risk of ischaemic stroke. If an arrhythmia goes undetected, the patient may not receive the appropriate treatment, which increases the likelihood of a recurrent stroke.
The technology developed and continuously improved by KTU researchers and Teltonika Telemedic enables continuous heart rhythm monitoring. When a suspicious episode is detected, the system alerts the patient via an on-screen notification or vibration. This allows the event to be captured precisely when it occurs rather than days or weeks later during a doctor's appointment.
It does not require adhesive electrodes or additional wires - the user simply touches the electrodes integrated into the device. Within about a minute, a more detailed ECG recording is made, showing the heart's electrical activity from several directions, and the data are then sent to the doctor.
In addition, the system will analyse not only the fact that an arrhythmia has occurred. It will use an arrhythmia aggregation parameter showing how episodes of rhythm disturbance are distributed over time - whether they occur evenly throughout the monitoring period or cluster into groups of short episodes.
"For a doctor, this kind of information would provide significantly more value than the mere fact that an arrhythmia was detected," says Marozas.
According to the KTU professor, such assessment makes it possible to monitor disease progression and assess a rising risk of complications at an earlier stage.
Patent marks a step towards wider application
To ensure that the data are reliable in real-life conditions, a specialised signal processing algorithm has also been integrated into the system. Movement, changes in body position or physical activity can distort signals, so the system first assesses their quality and only then sends suitable segments for more detailed analysis.
"The system distinguishes dangerous arrhythmias from noise caused by everyday activity using a multi-stage signal analysis process," says Marozas.
The patent granted to the system confirmed its technological novelty and became an important step towards further clinical trials and wider application. According to Marozas, the patent covers not only the wrist-worn device itself, but also arrhythmia analysis algorithms, arrhythmia aggregation and other new assessment parameters.
"The patent obtained is an important recognition of many years of interdisciplinary work and technological novelty. However, the greatest motivation remains saving patients' lives, improving their quality of life and advancing medicine in a way that benefits society," says Marozas.
A large interdisciplinary team contributed to both the patent application and the development of the technology, including KTU researcher Andrius Petrėnas, cardiologist Justinas Bacevičius from Vilnius University Hospital Santaros Clinics, KTU researchers Andrius Sološenko, Saulius Daukantas and Monika Butkuvienė, as well as KTU doctoral students, engineers, and medical residents from Vilnius University Hospital Santaros Clinics. For patients, the "TeltoHeart" device primarily means a simpler route to a doctor's consultation. According to Ilgevičius, if a person is recovering at home after surgery or a serious illness, they do not necessarily need to travel to a healthcare facility for tests.
The solution can now be implemented in the majority of healthcare institutions across Lithuania. Patients can use the solution together with a physician's consultation, while telemedicine services are already available in some outpatient clinics through projects funded by the European Union.
Nevertheless, broader reimbursement of such solutions for at-risk patients, such as people who have suffered a stroke, remains a future objective. According to the technology's developers, reimbursement policies may ultimately determine whether advanced remote monitoring solutions become widely accessible to the patients who need them most.
10 foods that influence stroke risk - MSN
Instead get EXACT DIET PROTOCOLS from your doctor and dietician. You don't want to do this wrong, do you?
10 Foods That Influence Stroke RiskCertain foods can raise the likelihood of stroke by increasing blood pressure, cholesterol, or contributing to vascular damage. Items such as processed meats, sugary drinks, fried foods, red meat, and salty snacks are linked to higher risk, while olive oil, garlic, berries, citrus fruits, and yogurt may help protect vascular health. Understanding these dietary effects can support better choices for stroke prevention. References 10 foods that can cause a stroke & 10 foods that minimize the risk | The Spicy Chefs10 Foods to Avoid After Having a Stroke | Flint Rehab | flintrehab.com 10 Foods That Increase Stroke Risk and 10 That Protect Against It | school-of-life.net What Are the Worst Foods for Your Brain? - WebMD | webmd.com Foods That Help Prevent Stroke - Mass General Brigham | massgeneralbrigham.org 5 Foods that Prevent Stroke - Healthline | healthline.com Dietitians Reveal the One Dairy Pick That Could Cut Your Stroke Risk | EatingWell |
Tuesday, June 30, 2026
Blood metabolites reveal lifestyle links to brain health before dementia
Is your competent? doctor testing for these post stroke so those EXACT DEMENTIA PREVENTION PROTOCOLS CAN BE INITIATED? Sorry, nothing exists!
Your risk of dementia, has your doctor
told you of this? Your doctor is responsible for preventing this! Is
s/he willing to prevent this?
1. A documented 33% dementia chance post-stroke from an Australian study? May 2012.
2. Then this study came out and seems to have a range from 17-66%. December 2013.`
3. A 20% chance in this research. July 2013.
4. Dementia Risk Doubled in Patients Following Stroke September 2018
Blood metabolites reveal lifestyle links to brain health before dementia
A large analysis suggests that blood metabolites may capture how smoking, BMI, medication use, gut microbes, and other exposures intersect with cognition and brain structure years before dementia emerges.
In a recent study published in the journal Nature Aging, researchers investigated the relationships between blood metabolites, magnetic resonance imaging (MRI) measures, and general cognition in middle-aged adults without dementia.
Alterations in peripheral metabolism have been increasingly implicated in brain diseases, including Alzheimer’s disease (AD). Studies have linked blood metabolites, such as lipids, lipoproteins, and amino acids, to AD-related phenotypes. Functional and structural changes in the brain, as well as changes in neuropsychological markers, appear years before AD manifestation. Specifically, brain atrophy and white matter hyperintensities are neurodegenerative and vascular markers associated with AD.
Studying early metabolic changes related to these features may help identify metabolites critical to disease etiology. Besides genetics, the exposome (lifestyle factors, medication use, and other environmental exposures) and the gut microbiome can influence metabolite levels, and their management offers opportunities to stabilize metabolism and counter metabolic changes associated with disease.
The study and findings
In the present study, researchers evaluated the association of blood metabolites with general cognition and MRI markers in dementia-free middle-aged adults. First, plasma levels of 1,387 metabolites were measured in 1,082 participants of the Rotterdam study. Next, the team assessed the cross-sectional relationships of 991 frequent metabolites with general cognition and MRI markers in 1,068 participants after data preprocessing. This revealed significant associations between 14 metabolites and cognition.
Increased levels of uridine, 2-deoxyuridine, ergothioneine, and two uncharacterized metabolites, and reduced levels of seven sulfated xenobiotics and two uncharacterized metabolites were associated with improved cognition. After adjustment for education, 12 of these 14 associations remained significant; after further adjustment for smoking, diabetes, and hypertension, all 14 remained nominally associated, but no longer met FDR-adjusted significance.
Regarding MRI markers, S-adenosylhomocysteine was associated with white matter lesion (WML) volume, while 21 metabolites were associated with total brain volume. No individual metabolite was significantly associated with hippocampal volume after multiple-testing correction.
Higher levels of 6-bromotryptophan, glycerophosphorylcholine, argininate, X-11787, and three sphingomyelins were associated with increased brain volume. Meanwhile, lower levels of N-lactoyltyrosine, six metabolites related to caffeine degradation, and seven intercorrelated metabolites, including hydroxylated acylcarnitines and a hydroxylated dicarboxylic fatty acid, were associated with elevated brain volume. The team also conducted a replication analysis in an independent sample of 847 older adults from the Rotterdam study. In this cohort, nine of 14 metabolites were significantly associated with cognition.
A second replication analysis was conducted in the Alzheimer Gut Microbiome Project (AGMP) cohort of 512 participants; associations with three cognitive scores were investigated: the Uniform Data Set (UDS) Benson Figure Total Delayed score, the National Alzheimer’s Coordinating Center (NACC) Montreal Cognitive Assessment (MoCA) score, and the Craft Story Delayed Recall (CRAFTDRE) score. Seven metabolites were significantly associated with at least one cognitive score. Across the RSI-4 and AGMP replication analyses, all 14 cognition-associated metabolites were significant in at least one replication test.
The researchers also compared the metabolite signature of cognition with incident AD in the older RSI-4 cohort, finding strong concordance between metabolites linked to cognition and those linked to later AD diagnosis.
Sex-stratified analyses revealed suggestive evidence of interactions with sex for multiple metabolites associated with MRI markers and general cognition. In particular, reduced levels of five metabolites, including glycocholate and taurocholate, were significantly associated with improved cognition in females only. Conversely, higher N-acetyl-aspartyl-glutamate was associated with improved cognition in males only. However, these sex-stratified findings were exploratory and did not replicate in the older RSI-4 cohort.
Next, the team investigated how much variance of metabolites was explained by genetic, lifestyle, microbial, clinical, and medication features. Among cognition-related metabolites, lifestyle features explained a substantial part of the variance of nine metabolites. Likewise, clinical, medication, and lifestyle features were the major factors affecting the variance of metabolites associated with WML or total brain volume.
The researchers also assessed associations of cognition- and MRI marker-related metabolites with clinical features, lifestyle factors, gut microbiota, and medication use using regression analyses. This revealed a significant association of 13 (out of 14) cognition-related metabolites with distinct lifestyle factors. Specifically, smoking was associated with increased levels of sulfated metabolites and lower levels of 2′-deoxyuridine and uridine.
In addition, observationally increased ergothioneine levels, linked to improved cognition, were associated with lower body mass index (BMI), higher alcohol intake, and higher education. Meanwhile, lower ergothioneine levels were associated with antacid use. Among medications and clinical factors, antidiabetic medication and diabetes showed the most associations. BMI and alcohol intake showed the most associations with metabolites linked to MRI markers.
In particular, 20 metabolites linked to MRI markers were associated with alcohol intake or BMI. Among metabolites associated with MRI markers and cognition, 22 were significantly associated with specific gut microbiota. For instance, higher ergothioneine was associated with increased abundance of 12 microbial genera, including six genera that showed positive associations, such as Fusicatenibacter and Romboutsia. Mediation analyses revealed that ergothioneine mediated 31.5% of the negative association between antacid use and cognition in an exploratory cross-sectional mediation analysis. The authors cautioned that this does not establish causality and that confirmation is required in longitudinal and mechanistic studies before any clinical prescribing implications can be drawn.
Conclusions
Taken together, the findings suggest that lifestyle factors play a substantial role in shaping blood metabolites linked to cognition and MRI markers in middle-aged people without dementia. Smoking was identified as a crucial lifestyle factor associated with cognition-associated metabolites. Meanwhile, BMI, antidiabetic medication, diabetes, and alcohol intake were associated with metabolites linked to MRI markers. Overall, the results could inform future prevention and intervention efforts, but they do not prove that modifying any single factor will improve brain outcomes.
Want to read later? Download your PDF copy by clicking here.
Journal reference:
- Ahmad S, Wu T, Arnold M, et al. (2026). The blood metabolome of brain health in midlife and influences of genes, microbiome and exposome. Nature Aging. DOI: 10.1038/s43587-026-01149-4, https://www.nature.com/articles/s43587-026-01149-4
Friday, May 29, 2026
Coconut Oil
Your competent? doctor can summarize how coconut oil can help or just say it is poison, your choice of what to believe.
Ask your doctor to clarify with valid research points.
The Hype About Coconut Oil Benefit Is False
A Harvard professor just busted the myth that coconut oil is good for you, calling it 'pure poison'
I will still use it, reasons found in these posts.
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coconut oil
(65 posts)
Coconut Oil
Coconut oil, derived from the dried Cocos nucifera (coconut) fruit, is made up of 90% saturated triglycerides and is used as a skin- and hair-conditioning agent as well as a fragrance ingredient in hundreds of formulations at concentrations ranging from 0.0001% to 70%. Notably, coconut oil also has a long history of traditional medicine use in China and India and in food preparation. Indeed, coconut oil is a staple ingredient in the Sri Lankan diet as well as others in South Asia, Africa, and South America, where coconut is cultivated.
Coconut oil has also been used for skin moisturizing for several centuries among people living in tropical and subtropical regions. It is known to exhibit antiaging, anti-inflammatory, and antioxidant activity. This column focuses on recent evidence of wide-ranging benefits to skin health from the use of coconut oil.
Chemical Composition
Virgin coconut oil, or the unprocessed oil derived from a fresh coconut endocarp, is composed mainly of saturated fatty acids, including capric (7%), caprylic (8%), gallic, lauric (49%), linoleic (2%), myristic (18%), oleic (6%), palmitic (8%), stearic (2%), and phenolic acids, as well as medium-chain triglycerides, polyphenols, iron, and vitamins E and K. Coconut oil is known to exhibit anticancer , antidiabetic, anti-inflammatory, antimicrobial, antioxidant, hypocholesterolemic, and skin moisturizing activity.
The monoglyceride monolaurin, derived from lauric acid, makes up 50% of the fat content of coconut and exhibits notable antibacterial effects against Propionibacterium acnes, Staphylococcus aureus, and Staphylococcus epidermidis. Coconut oil in concentrations of 5%-40% (w/w) displays antibacterial activity against Pseudomonas aeruginosa, Escherichia coli, Proteus vulgaris, and Bacillus subtilis.
Wound Healing
In 2010, Nevin and Rajamohan demonstrated that topical application of cold-pressed virgin coconut oil to young female Sprague-Dawley rats once daily for 10 days yielded more rapid epithelialization and healing in treated rats vs controls. Increased levels of collagen, elastin, glycosaminoglycans, hexose, proteins, and sialic acid in the granulation tissue were observed.
Nine years later, Meliala and colleagues showed that virgin coconut oil and hydrolyzed virgin coconut oil enhanced protein expressions of MMP-9, PDGF-BB, and TGF-beta-1 in NIH-3T3 cell lines, suggesting their activity in wound healing. They noted that the hydrolyzed version was more active.
Hair Health
Virgin coconut oil has been reported to provide emollient and hair-conditioning effects, deliver shine, diminish hair breakage, and darken hair. It also contributes to the treatment of hair loss caused by reputed antioxidant, anti-inflammatory, antimicrobial, and immunomodulatory activity.
Coconut oil is a key ingredient in shampoos, conditioners, creams, and ointments, in which it is used to strengthen damaged and undamaged hair. In a 2022 systematic review of the literature on oils used traditionally to treat the hair of people of color, Phong and colleagues identified 22 articles meeting inclusion criteria. Based on this material, they concluded that coconut oil has demonstrated clinical efficacy in treating brittle hair and hair infestations.
Anti-Inflammatory Activity and Skin Barrier Health
By promoting expression of cornified envelope constituents in the epidermis, coconut oil improves the barrier function of the stratum corneum.
In 2017, Kim and colleagues studied the effects of cultured coconut extract on human explanted skin exposed to ultraviolet B radiation. Samples of skin treated with the extract evinced elevated expression of cornified cell envelope components, which aid the stratum corneum in providing skin protection. Inflammatory marker expression was also diminished in the treated group. Furthermore, collagen and hyaluronan synthase-3 expression were increased in the treated group. The authors concluded that cultured coconut extract improved barrier functions of the stratum corneum and delivered anti-inflammatory activity — roles they ascribed to the abundant polyphenols and fatty acids found in coconut.
In 2018, Varma and colleagues demonstrated that virgin coconut oil exhibits anti-inflammatory and skin-protective activity by inhibiting inflammatory markers and ameliorating skin barrier function, specifically elevating the expression of Aquaporin-3, involucrin, and filaggrin. They concluded that these roles justify the use of virgin coconut oil in skin care products.
Compared to Other Oils
Used as a moisturizer to treat mild-to-moderate dry skin, coconut oil has demonstrated comparable efficacy and safety to mineral oil.
In a review of clinical studies conducted by Poljšak and Kocevar Glavac in 2022, coconut oil was among three oils used in patients with atopic dermatitis and outperformed mineral oil. Coconut oil also significantly diminished S aureus colonization compared with olive oil in another trial.
Treating Newborn Skin
In 2014, Evangelista and colleagues conducted a randomized, double-blind clinical trial to compare the effects of topical virgin coconut oil and mineral oil on SCORAD (SCORing of Atopic Dermatitis) values, transepidermal water loss (TEWL), and skin capacitance in 117 pediatric patients with mild-to-moderate atopic dermatitis over 8 weeks. The coconut oil group performed better along every metric, with both oils diminishing the signs of atopic dermatitis. The investigators concluded that topical virgin coconut oil was superior to mineral oil for pediatric patients with mild-to-moderate atopic dermatitis in terms of clinical and instrumental evaluations.
The next year, Nangia and colleagues determined that coconut oil application decreased TEWL without elevating skin colonization in very low birthweight neonates.
In 2018, Strunk and colleagues conducted an open-label, randomized controlled trial to assess the viability, safety, and effectiveness of topical coconut oil to treat the skin of 72 very preterm infants (< 30 weeks’ gestation). No adverse effects emerged from the study, which found that topically applied coconut oil was a feasible treatment. The Neonatal Skin Condition Score in the coconut oil group was maintained throughout the study, whereas it decreased in the control group. The investigators concluded that the superiority of coconut oil over routine care warrants additional study.
Two years later, Konar and colleagues reported on a randomized controlled trial to ascertain the efficacy of coconut oil application on the skin of preterm infants. The intervention group received virgin coconut oil (n = 1146), and the control group received only body massage (n = 1148). Greater skin maturity as well as neurodevelopmental results were observed in the coconut oil group, along with fewer occurrences of hypothermia and apnea. No significant adverse side effects were observed.
Safety
The final report on the safety evaluation of coconut oil, published in 2011, revealed that the oil and its related constituents are considered safe as cosmetic ingredients by the Cosmetic Ingredient Review Expert Panel.
Conclusion
Coconut oil has a lengthy history of medical, nutritional, and other uses. Current evidence supports the use of this botanical agent to promote skin health. More research is needed to ascertain more varied and curative applications for this popular natural product.
Dr Baumann is a private-practice dermatologist, researcher, author, and entrepreneur in Miami, and a cosmetic dermatologist for Body+Beauty Labs. She founded the Division of Cosmetic Dermatology at the University of Miami in 1997. The third edition of her bestselling textbook, Cosmetic Dermatology, was published in 2022.
Wednesday, May 27, 2026
Join us FREE for a live webinar focused on neuroplasticity, motor learning, and functional recovery June 15th at 8 pm EST Register Here In this free live webinar, you’ll learn how to intentionally drive brain reorganization through task specific, evidence-based interventions that improve movement, engagement, and long term outcomes across neurologic populations. Tapping Neuroplasticity to Enhance Neurological Recovery Monday June 15th 8 pm - 9 pm EST Save Your Seat For FREE Register today and start transforming the way you approach neurological rehab! CUSTOMER SERVICE: Mon-Fri, 7AM-6PM Central Phone: (800) 433-9570 | Fax: (615) 376-8233 | Email Us | FAQs FOLLOW US: Facebook Instagram LinkedIn YouTube © Summit Professional Education | 501 Union St Ste 545 PMB 626120, Nashville, TN 37219-1876 Unsubscribe | Manage Preferences
Completely useless; they know ABSOLUTELY NOTHING THAT WILL GUARANTEE NEUROPLASTICITY!
You haven't identified the EXACT signals between neurons that tells one neuron to drop their use and take on a neighboring neuron's use! That could then make neuroplasticity repeatable on demand. Until that occurs ALL OF THIS SUPPOSED NEUROPLASTICITY RESEARCH IS ALMOST COMPLETELY FUCKING USELESS!
In this free live webinar, you’ll learn how to intentionally drive brain reorganization through task specific, evidence-based interventions that improve movement, engagement, and long term outcomes across neurologic populations.
Tapping Neuroplasticity to Enhance Neurological Recovery Monday June 15th 8 pm - 9 pm EST |
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Sunday, May 17, 2026
How to Recover Hand From Stroke — Effortless Exercise Automatic Hand Trainer Gloves
Ask your doctor for the EXACT RESEARCH PROVING THIS WORKS!
How to Recover Hand From Stroke — Effortless Exercise Automatic Hand Trainer Gloves
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