Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Wednesday, December 14, 2016

Dietary magnesium intake and the risk of cardiovascular disease, type 2 diabetes, and all-cause mortality: A dose-response meta-analysis of prospective cohort studies

Useless information unless the full article gives the foods used. I.E. a diet protocol. This is so fucking simple, create protocols for every intervention. Who cares if they haven't been totally proven out? Update them as more information comes in. But the perfect is better than the good enough, so don't expect anything useful from the current mindset for 50 years.
https://www.mdlinx.com/internal-medicine/medical-news-article/2016/12/14/magnesium-cardiovascular-disease-type-2-diabetes/6975741/?
BMC Medicine, 12/14/2016
For this study, researchers directed a dose–response meta–analysis of prospective cohort studies keeping in mind the end goal to research the relationship between's magnesium consumption and the risk of cardiovascular disease (CVD), type 2 diabetes (T2D), and all–cause mortality. Expanding dietary magnesium consumption is connected with a diminished risk of stroke, heart failure, diabetes, and all–cause mortality, but not CHD or total CVD. These discoveries bolster the idea that expanding dietary magnesium may give health benefits.

Methods

  • In this study researchers searched PubMed, EMBASE, and Web of Science for articles.
  • They searched articles that contained risk estimates for the outcomes of interest and were published through May 31, 2016.
  • The pooled results were examined utilizing a random–effects model.

Results

  • 40 prospective cohort studies totaling more than 1 million participants were incorporated into the examination.
  • Amid the follow–up periods (ranging from 4 to 30 years), 7678 cases of CVD, 6845 cases of coronary heart disease (CHD), 701 cases of heart failure, 14,755 cases of stroke, 26,299 cases of T2D, and 10,983 deaths were reported.
  • No significant affiliation was seen between expanding dietary magnesium consumption (per 100 mg/day increment) and the risk of total CVD (RR: 0.99; 95% CI, 0.88–1.10) or CHD (RR: 0.92; 95% CI, 0.85–1.01).
  • However, the same incremental increase in magnesium consumption was connected with a 22% diminishment in the risk of heart failure (RR: 0.78; 95% CI, 0.69–0.89) and a 7% reduction in the risk of stroke (RR: 0.93; 95% CI, 0.89–0.97).
  • Besides, the outline relative risks of T2D and mortality per 100 mg/day increment in magnesium consumption were 0.81 (95% CI, 0.77–0.86) and 0.90 (95% CI, 0.81–0.99), respectively.
Go to PubMed Go to Abstract Print Article Summary Cat 2 CME Report

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