Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Tuesday, February 1, 2022

Serum glucose may help identify those admitted for stroke at greatest risk for recurrence

 What is the name of the test we need to be asking our doctors to perform? And the readout that is a concern? Since doctors and hospitals don't read and implement research findings it is up to stroke survivors to fill in the gaps.

Serum glucose may help identify those admitted for stroke at greatest risk for recurrence

Presence of hyperglycemia at hospital admission for stroke was linked with elevated risk for subsequent stroke by 90 days, an increase that dual antiplatelet therapy seemingly failed to curtail, researchers reported.

According to research published in the Journal of the American Heart Association, serum glucose measurement may represent a quick and easy assay to identify patients hospitalized for transient ischemic attack or minor ischemic stroke who are at particularly high risk for subsequent stroke.

diabetes glucose test strip
Source: Adobe Stock

“The risk of subsequent stroke is as high as 17% in the 90 days following the index event, but this risk is front-loaded within the first 7 days. For this reason, there is a need to incorporate dynamic physiological metrics into risk stratification schemes, and not simply long-term risk factors,” Brian Mac Grory, MB BCh BAO, MRCP, assistant professor of neurology at Duke University School of Medicine, and colleagues wrote. “Serum glucose is an intriguing potential predictor of recurrent stroke risk, because it is already assessed in the majority of patients with acute stroke using widely available, low-cost assays.”

Researchers conducted a secondary analysis of the POINT trial to assess the relationship between hyperglycemia ( 180 mg/dL) compared with normoglycemia (< 180 mg/dL) and 90-day outcomes following stroke hospitalization. Researchers also evaluated the effects of DAPT in this population. The primary endpoint was subsequent ischemic stroke.

POINT was a randomized controlled trial that assessed the effects of aspirin plus clopidogrel compared with aspirin alone for prevention of recurrent stroke among patients with TIA or minor ischemic stroke.

As Healio previously reported, although DAPT may lower risk for recurrent stroke compared with aspirin alone, it may increase risk for major hemorrhagic bleeding.

Impact of hyperglycemia on secondary stroke risk

Among the 4,878 participants in the POINT study, by 90 days, 267 experienced recurrent stroke, with a cumulative incidence of 9.7% in patients with hyperglycemia and 5.2% in those normoglycemic (log-rank P < .001).

After adjusting for age, sex, race, ethnicity, treatment assignment, index event classification and vascular risk factors as covariates, researchers observed significant association between hyperglycemia at hospital admission for TIA or minor ischemic stroke and risk for subsequent stroke compared with normoglycemia (HR = 1.5; 95% CI, 1.05-2.14; P = .01).

According to the study, hyperglycemia at stroke admission was also tied to a composite of ischemic stroke, MI or vascular death compared with normoglycemia (HR = 1.55; 95% CI, 1.1-2.2; P = .01).

In a similarly adjusted model, researchers found no association between hyperglycemia and major hemorrhage compared with normoglycemia (HR = 0.47; 95% CI, 0.11-1.99; P = .31).

DAPT in hyperglycemia following stroke

Researchers reported no risk reduction for subsequent stroke with DAPT initiation in patients with hyperglycemia (HR = 1.18; 95% CI, 0.69-2.03), but found there was lower risk in patients with normoglycemia (HR = 0.63; 95% CI, 0.48-0.83; P for interaction = .04).

“The benefits of clopidogrel/aspirin were not apparent in the small subgroup of patients with hyperglycemia, with an interaction observed between clopidogrel and serum glucose on subsequent stroke,” the researchers wrote.

In a sensitivity analysis that incorporated serum glucose as a continuous variable, researchers saw evidence of a nonlinear relationship between serum glucose and risk for subsequent stroke (P < .001).

 

No comments:

Post a Comment