Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Thursday, February 24, 2022

B-vitamin supplementation on mitigating post-stroke cognition and neuropsychiatric sequalae: A randomized controlled trial.

No benefit.

B-vitamin supplementation on mitigating post-stroke cognition and neuropsychiatric sequalae: A randomized controlled trial.

First Published February 23, 2022 Research Article 

Background and Purpose

A third of stroke patients suffer from post-stroke cognitive decline, depressive, and anxiety symptoms. B-vitamin supplementation provides a possible safe and affordable treatment to mitigate post-stroke neuropsychiatric sequelae via reducing homocysteine levels. Our study aims to examine the effect of B-vitamin supplementation in the prevention of post-stroke cognitive decline, depressive, and anxiety symptoms. Our secondary aims were to investigate associations between baseline factors and the three outcomes.

Methods

Patients were recruited as part of a Singaporean sub-study of a randomized controlled trial that examined the effect of B-vitamin supplementation on recurrent cardiovascular events. Cognitive decline, depressive and anxiety symptoms were assessed with neuropsychological assessments and Hospital Anxiety and Depression Scale 6 monthly. Cox regression analyses was performed to determine treatment efficacy. Logistic regression used to examine factors associated with cognitive decline, depressive and anxiety symptoms.

Results

A total of 707 were included in the analyses. Survival and hazards ratio analysis showed no treatment effect of B-vitamins on cognitive decline, depressive and anxiety symptoms. Cognitive decline was only associated with age. Depressive symptoms were associated with large anterior cerebral infarcts and hyperlipidaemia

Conclusions

Our study showed no benefit of supplementation with B-vitamins for post-stroke cognitive decline, depressive or anxiety symptoms. Depressive symptoms was associated with larger anterior cerebral infarcts, which may be reflective of the disability associated with larger infarcts.

Registration-URL: http://www.clinicaltrials.gov; Unique identifier: NCT00097669

 

No comments:

Post a Comment