Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Wednesday, February 1, 2023

Early neurological deterioration in patients with acute ischemic stroke: a prospective multicenter cohort study

Leaders would put into action the research needed to prevent early neurological deterioration. But you're not leaders; ARE YOU? 

So you described a problem, offered no solution, didn't suggest further research; USELESS. And didn't even consider that early neurological deterioration might be caused by all the neurons dying because you've done nothing to stop the 5 causes of the neuronal cascade of death in the first days.

 

Early neurological deterioration in patients with acute ischemic stroke: a prospective multicenter cohort study

Abstract

Background:

There is still no precise knowledge of the causes of progression in patients with acute ischemic stroke (AIS), and we are unable to predict patients at risk.

Objective:

To explore the frequency, predictive factors, and the prognosis of early neurological deterioration (END) in patients with AIS

Methods:

In this prospective multicenter observational study, we assessed patients with AIS admitted to 18 hospitals in Henan, China. We defined END as an increase of ⩾2 points in total National Institutes of Health Stroke Scale (NIHSS) score or ⩾1 point in the motor items of the NIHSS within 7 days after admission. Risk factors were analyzed using multivariate logistic regression models. Prognosis was evaluated using the modified Rankin Scale (mRS), with poor prognosis defined as mRS 3–6.

Results:

A total of 9114 patients with AIS within 24 h of symptom onset were enrolled in the study. END occurred in 1286 (14.1%) patients. The highest incidence (62.5%) of END occurred within 24 h after admission. After adjusting potential confounders, age, body mass index, waist–hip ratio, systolic blood pressure, baseline NIHSS, disabled at baseline, history of atrial fibrillation, diabetes mellitus, intracranial arterial stenosis, infarct location in the lenticulostriate artery area and cerebral watershed, neutrophils, lymphocytes, uric acid, and triglycerides were identified as independent predictors for END. END was significantly associated with poor prognosis at 90 days, and the adjusted OR was 1.74 (95% CI: 1.53–1.97).

Conclusion:

One in seven hospitalized patients with AIS may experience END within 24 h of onset. The highest incidence of END occurred within 24 h of admission and decreased steeply with time. Easily identifiable risk factors predict END and could help understand the causal mechanisms and thereby prevent END.

Introduction

Early neurological deterioration (END) usually refers to the decline in neurological function that occurs within a few hours or days of the onset of acute ischemic stroke (AIS).1 Studies have reported END frequencies ranging from 5% to 40%, which could have resulted from different evaluation methods and inclusion criteria.2,3 Previous studies have indicated that END may affect the prognosis of patients with AIS.4 However, the etiology and pathogenesis of END in AIS are complex, and clear descriptions, accurate and reliable early prediction indicators, and effective prevention and treatment strategies are lacking.5 Therefore, it is important to explore END development in patients with AIS.
Although previous studies have reported on AIS with END, most were retrospective and focused on specific populations with stroke, such as patients receiving intravenous thrombolysis and endovascular therapy or patients with mild stroke.68 In addition, most studies were single-center, small-sample studies; therefore, large multicenter prospective cohort data on END are lacking. We aimed to improve END awareness by exploring the frequency, risk factors, and functional outcomes in patients with AIS using multicenter stroke registry data in Henan, China.

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