Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Monday, April 3, 2023

Locked-in syndrome revisited

Are your doctors well versed in identifying and treating this?  You better ask now while you still have the ability to talk.

Locked-in syndrome revisited


Abstract

The locked-in syndrome (LiS) is characterized by quadriplegia with preserved vertical eye and eyelid movements and retained cognitive abilities. Subcategorization, aetiologies and the anatomical foundation of LiS are discussed. The damage of different structures in the pons, mesencephalon and thalamus are attributed to symptoms of classical, complete and incomplete LiS and the locked-in plus syndrome, which is characterized by additional impairments of consciousness, making the clinical distinction to other chronic disorders of consciousness at times difficult. Other differential diagnoses are cognitive motor dissociation (CMD) and akinetic mutism. Treatment options are reviewed and an early, interdisciplinary and aggressive approach, including the provision of psychological support and coping strategies is favoured. The establishment of communication is a main goal of rehabilitation. Finally, the quality of life of LiS patients and ethical implications are considered. While patients with LiS report a high quality of life and well-being, medical professionals and caregivers have largely pessimistic perceptions. The negative view on life with LiS must be overthought and the autonomy and dignity of LiS patients prioritized. Knowledge has to be disseminated, diagnostics accelerated and technical support system development promoted. More well-designed research but also more awareness of the needs of LiS patients and their perception as individual persons is needed to enable a life with LiS that is worth living.

Introduction

The locked-in syndrome (LiS) was first described by Plum and Posner 1972 and is one of the most disabling entities. Patients are locked in their own bodies, unable to move due to quadrioplegia, except for vertical eye and eyelid movements. Although no other voluntary muscle movements are possible, cognitive abilities of the patients are not impaired.1
In 1979, Bauer et al. introduced further subcategorizations: he distinguished the classical LiS from the complete and incomplete LiS to describe patients whose capacity of voluntary movements went below or above the classical LiS motor symptoms. In complete LiS, no voluntary movements are achievable at all, and patients lack any possibility to communicate directly, which makes further diagnostics indispensable. Incomplete LiS is a less severe form, which can also be a transitory stage of recovery, where patients are able to execute more voluntary movements than in the classical form of LiS.2 For patients who additionally suffer from disturbances of consciousness, the term locked-in plus (LiPS) was introduced, by the Salzburg Coma Group.3
Nevertheless, the first literary description of LiS was written long before these modern medical ones, by Alexandre Dumas in his novel The Count of Monte Cristo in 1844. Monsieur Noirtier de Villefort, a key character, is not able to speak, ‘motionless as a corpse, he greeted his children with bright, intelligent eyes . . . ’. Dumas even showed that he understood the basic aetiology of LiS as he describes strokes and brain bleeds in his story.4 As a metaphor for human existence, LiS can be found throughout fictional literature.5 As Kondziella6 shows, even complete LiS was described long before the medical community became aware of it by Roald Dahl in a short story William and Mary (1959).
One of the most influencing non-fictional books on the topic is The Diving Bell and the Butterfly written by Jean-Dominique Bauby in 1997 by blinking. The author, himself suffering from LiS, gives very personal and touching insights into the life of a LiS patient and shows the pain but also beautiful moments of his existence in this autobiography.7
LiS is a very rare syndrome and no exact incidence or prevalence is known. In 1986, Patterson and Grabois obtained a sample of 139 cases including six own cases and cases from a literature review. They found a mean age of onset of 52 years (range: 16–90) with more males than females affected (85 to 52). A total of 89 patients were diagnosed with classical, 46 incomplete and 3 complete LiS (one unclassified); 10 patients showed transient states. Eighty-two patients suffered an infarction of the pons, other vascular and non-vascular etiologies being less common. Roughly one third of the patients had a history of hypertension. A mortality rate of 60% was reported, but no time frame given, although the majority of deaths occurred within the first 4 months after onset.8 A newer study reports a mortality rate of 75% in the acute phase.9 The most reported causes of death are pulmonary complications and further brainstem damage.8,9 Sensory perception varied wildly in the population from normal to absent. It was also shown that substantial recovery is possible8,1016 and can continue over years,17 possibly by reorganization of the descending pathways.18 One survey of chronic LiS patients showed a recovery of functional movements in 72%.19 Distal motor functions are more likely to recover,20 and even when large brainstem strokes are apparent in magnetic resonance imaging (MRI), substantial recovery is possible.21
In 2013, a study estimated a prevalence of 0.7/10,000 patients with classical LiS in Dutch nursing homes.22 ALIS (Association of Locked-in Syndrome) in France states on its website that more than 500 French people live in LiS in 2022.23 Life expectancy has improved in the last decades for LiS patients,24 some living for many decades in LiS.25
The aim of this review is to give an up-to date overview of LiS, focusing on anatomy, pathophysiology, treatments, quality of life, ethics and new developments in the field. Not only are the structures that can be affected in LiS and the resulting symptoms discussed but also the anatomical foundations of LiPS. Moreover, differential diagnosis of LiS that have to be considered and treatment options are recapitulated. Finally, ethical considerations are presented. This review is important to inform and raise awareness about LiS and counteract prevalent misconceptions about this rare disease among medical doctors and other health care staff.

No comments:

Post a Comment