Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Tuesday, August 15, 2023

Impairments of the ipsilesional upper-extremity in the first 6-months post-stroke

ABSOLUTELY FUCKING USELESS. Describing impairments and not providing any protocols to fix those impairments is your first step to getting fired! Survivors would like recovery protocols. GET THERE!

Impairments of the ipsilesional upper-extremity in the first 6-months post-stroke

Abstract

Background

Ipsilesional motor impairments of the arm are common after stroke. Previous studies have suggested that severity of contralesional arm impairment and/or hemisphere of lesion may predict the severity of ipsilesional arm impairments. Historically, these impairments have been assessed using clinical scales, which are less sensitive than robot-based measures of sensorimotor performance. Therefore, the objective of this study was to characterize progression of ipsilesional arm motor impairments using a robot-based assessment of motor function over the first 6-months post-stroke and quantify their relationship to (1) contralesional arm impairment severity and (2) stroke-lesioned hemisphere.

Methods

A total of 106 participants with first-time, unilateral stroke completed a unilateral assessment of arm motor impairment (visually guided reaching task) using the Kinarm Exoskeleton. Participants completed the assessment along with a battery of clinical measures with both ipsilesional and contralesional arms at 1-, 6-, 12-, and 26-weeks post-stroke.

Results

Robotic assessment of arm motor function revealed a higher incidence of ipsilesional arm impairment than clinical measures immediately post-stroke. The incidence of ipsilesional arm impairments decreased from 47 to 14% across the study period. Kolmogorov–Smirnov tests revealed that ipsilesional arm impairment severity, as measured by our task, was not related to which hemisphere was lesioned. The severity of ipsilesional arm impairments was variable but displayed moderate significant relationships to contralesional arm impairment severity with some robot-based parameters.

Conclusions

Ipsilesional arm impairments were variable. They displayed relationships of varying strength with contralesional impairments and were not well predicted by lesioned hemisphere. With standard clinical care, 86% of ipsilesional impairments recovered by 6-months post-stroke.(86% is still failure!)

Introduction

Stroke is one of the leading causes of death and disability around the world, with over 13.7 million new cases each year [1]. Upper extremity motor impairment after stroke can be profound, impacting nearly 75% of survivors [2]. These impairments have traditionally been viewed as mainly affecting the contralesional limb, and as a result, recovery has primarily been gauged by contralesional arm motor performance [3,4,5]. Previous work has stressed the importance of analyzing ipsilesional impairments throughout stroke recovery [6, 7], and has demonstrated that up to 37% of stroke survivors experience motor impairments in their ipsilesional limb [8]. In cases where the contralesional limb is unable to recover sufficiently, the ipsilesional limb then becomes the dominant limb required to complete activities of daily living [9]. If the ipsilesional limb is also impaired, activities of daily living can become increasingly difficult to complete [10], and functional recovery could prove challenging [11].

A number of studies have examined ipsilesional arm impairments after stroke [8, 11,12,13,14]. While ipsilesional arm impairments seem to improve with time post-stroke [6, 15, 16], there are incongruent findings around the influence of the stroke-lesioned hemisphere on ipsilesional impairments. Some studies have suggested that left-hemisphere damage impacts movement trajectory direction and curvature, and right-hemisphere damage impacts movement endpoint control [14, 17,18,19]. As these studies only recruited right-hand dominant chronic stroke participants, the findings must be interpreted carefully, particularly in those with subacute stroke. Other studies have suggested no hemispheric differences are present in ipsilesional arm motor behaviour following stroke [6, 8, 20, 21]. This incongruency must be sorted out to provide a definitive answer as to if lesioned hemisphere impacts the severity and/or type of motor impairments observed in the ipsilesional arm, better informing clinical practice when prescribing rehabilitation for stroke survivors [18].

Another area of debate in the literature focuses on whether ipsilesional motor impairments scale with the severity of contralesional motor impairments. The current literature presents conflicting results, with a recent study finding that ipsilesional impairments scale with the severity of the contralesional impairments [18], yet another recent study suggesting that ipsilesional impairments are unrelated to contralesional impairment [6]. However, one of these studies examined behaviour in a smaller subacute stroke sample (n = 19) [6], and the other focused on chronic stroke [18]. Better understanding the relationship between the severity of ipsilesional and contralesional arm impairments is important, and a larger, longitudinal study is required to determine how this relationship changes.

The main goal of the present study was to characterize ipsilesional arm motor impairments throughout the first 6-months post-stroke. Here, we examined how motor impairments change from their initial presentation in the subacute phase at 1-week post stroke to the chronic phase at 6-months post-stroke in both the ipsilesional and contralesional arm. Second, we determined the proportion of participants with persistent ipsilesional and contralesional motor impairments at 6-months post-stroke. Third, we determined if ipsilesional arm motor impairments scaled with the severity of contralesional arm motor impairments. Last, we determined if the side of the stroke-lesioned hemisphere was related to the severity and type of motor impairments seen in the ipsilesional arm.

More at link.

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