Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Thursday, August 13, 2020

Usefulness of stent strut deformity during thrombectomy for predicting the stroke etiology in acute large artery occlusion

 I can see almost zero usefulness in stroke etiology(the cause). Survivors want to know: WHAT THE FUCK ARE YOU DOING TO CURE ME?

Usefulness of stent strut deformity during thrombectomy for predicting the stroke etiology in acute large artery occlusion

Highlights

Underlying intracranial atherosclerotic stenosis can be a cause of refractory occlusion after stent-based thrombectomy.

Defining the stroke etiology is important for the treatment of acute large vessel occlusion.

Full-length stent visibility during thrombectomy can provide information on the stroke etiology or the clot characteristics.

Abstract

Background

Stent retriever thrombectomy has been regarded as the standard treatment for acute intracranial large artery occlusion. As fast recanalization is the most important factor for favorable outcomes in patients with stroke, defining the etiology is important for the treatment of acute large vessel occlusion. We aimed to investigate whether full-length stent visibility during thrombectomy could provide information on the stroke etiology or the clot characteristics using the stent strut deformity during thrombectomy.

Materials and methods

Intra-arterial thrombectomy was performed on 46 patients with Trevo stent as the first endovascular thrombectomy device. Patients were assigned to the full expansion group or the stent deformity group based on the shape of the stent strut during the endovascular procedure. The presence of stent deformity during the procedure, underlying intracranial atherosclerotic stenosis (ICAS), and residual stenosis at the occlusion site on the final conventional angiography, follow-up magnetic resonance angiography, or computed tomography angiography were retrospectively assessed.

Results

Recanalization without underlying arterial stenosis at the occlusion site was observed in 92.9 % (26/28 patients) and 50.0 % (8/16 patients) of the full expansion and stent deformity groups (p = 0.002), respectively. A significantly lower proportion of patients in the full expansion group demonstrated ICAS-related occlusion compared with that in the stent deformity group (3.6 % vs. 43.8 %, respectively; p = 0.002).

Conclusions

The degree of stent expansion during thrombectomy in acute large artery occlusion can be a useful predictor of the stroke etiology and potentially helpful to the operator for endovascular treatment planning.

 

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