Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Saturday, July 25, 2015

Dietary flavonoid intake and cardiovascular risk: A population-based cohort study

What flavonoids is your doctor recommending? Does your doctor even know about this?
Flavonoid Content of U.S. Fruits, Vegetables, and Nuts

The latest here:


Dietary flavonoid intake and cardiovascular risk: A population-based cohort study


The primary objective was establishing if flavonoid intake was inversely associated with the cardiovascular (CV) risk evaluated after 12–year follow–up; the associations between flavonoid intake and CV incidence and mortality and all–cause mortality were also evaluated. Flavonoid intake was inversely associated with CV risk, CV non–fatal events and all–cause mortality in a cohort with a low consumption of soy, tea and cocoa, which are typically viewed as the foods responsible for flavonoid–related benefits.

Methods

  • In 2001–2003, a cohort of 1,658 individuals completed a validated food–frequency questionnaire.
  • Anthropometric, laboratory measurements, medical history and the vital status were collected at baseline and during 2014.
  • The CV risk was estimated with the Framingham risk score.

Results

  • Individuals with the lowest tertile of flavonoid intake showed a worse metabolic pattern and less healthy lifestyle habits.
  • The 2014 CV risk score and the increase in the risk score from baseline were significantly higher with the lowest intake of total and all subclasses of flavonoids, but isoflavones, in a multiple regression model.
  • During follow–up, 125 CV events and 220 deaths (84 of which due to CV causes) occurred.
  • CV non–fatal events were less frequent in individuals with higher flavonoid intake (HR = 0.64; 95%CI 0.42–1.00 and HR = 0.46; 95%CI 0.28–0.75 for the second and third tertiles, respectively) in Cox–regression models, after multiple adjustments.
  • All subclasses of flavonoids, but flavones and isoflavones, were inversely correlated with incident CV events, with HRs ranging from 0.42 (flavan–3–ols) to 0.56 (anthocyanidins).
  • Being in the third tertile of flavan–3–ols (HR = 0.68; 95% CI 0.48–0.96), anthocyanidins (HR = 0.66; 95% CI 0.46–0.95) and flavanones (HR = 0.59; 95% CI 0.40–0.85) was inversely associated with all–cause mortality.
  • Total and subclasses of flavonoids were not significantly associated with the risk of CV mortality.
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