Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Monday, August 26, 2024

Effects of four-week intranasal oxytocin administration on large-scale brain networks in older adults

I can't make heads or tails if this did something useful, so go ask your competent? doctor.  If useful your competent? doctor needs to get testing on us unhealthy stroke survivors.

Effects of four-week intranasal oxytocin administration on large-scale brain networks in older adults

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https://doi.org/10.1016/j.neuropharm.2024.110130
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Highlights

  • Chronic IN-OT reduced rs-FC of right insula and left MCC with salience network

  • Chronic IN-OT enhanced rs-FC of left mPFC with default mode network

  • Chronic IN-OT enhanced rs-FC of left thalamus with basal ganglia–thalamus network

  • No significant chronic IN-OT effects on between-network rs-FC

  • Pronounced IN-OT effects on network-wise rs-FC after chronic than acute IN-OT

Abstract

Oxytocin (OT) is a crucial modulator of social cognition and behavior. Previous work primarily examined effects of acute intranasal oxytocin administration (IN-OT) in younger males on isolated brain regions. Not well understood are (i) chronic IN-OT effects, (ii) in older adults, (iii) on large-scale brain networks, representative of OT’s wider-ranging brain mechanisms. To address these research gaps, 60 generally healthy older adults (mean age = 70.12 years, range = 55-83) were randomly assigned to self-administer either IN-OT or placebo twice daily via nasal spray over four weeks. Chronic IN-OT reduced resting-state functional connectivity (rs-FC) of both the right insula and the left middle cingulate cortex with the salience network but enhanced rs-FC of the left medial prefrontal cortex with the default mode network as well as the left thalamus with the basal ganglia–thalamus network. No significant chronic IN-OT effects were observed for between-network rs-FC. However, chronic IN-OT increased selective rs-FC of the basal ganglia–thalamus network with the salience network and the default mode network, indicative of more specialized, efficient communication between these networks. Directly comparing chronic vs. acute IN-OT, reduced rs-FC of the right insula with the salience network and between the default mode network and the basal ganglia–thalamus network, and greater selective rs-FC of the salience network with the default mode network and the basal ganglia–thalamus network, were more pronounced after chronic than acute IN-OT. Our results delineate the modulatory role of IN-OT on large-scale brain networks among older adults.

Introduction

The neuropeptide oxytocin (OT) acts as a neuromodulator on social cognition and social behavior (Meyer-Lindenberg et al., 2011). OT is synthesized in the hypothalamus and processed along axonal projections for posterior pituitary and dendritic release into extracellular space, resulting in both local action and diffusion to distant brain regions, such as the amygdala and insula (Meyer-Lindenberg et al., 2011). One of the most well-documented roles of OT is its involvement in maternal and infant bonding. Additionally, OT is implicated in numerous physiological effects, such as reducing free cortisol levels, lowering blood pressure, inducing analgesia, and promoting wound healing (IsHak et al., 2011).

OT can be administered via diverse routes including intranasal, oral, and intravenous (Phung et al., 2021; Zhuang et al., 2022), but intranasal OT administration (IN-OT) circumvents the blood-brain barrier and is well-suited for the investigation of OT effects on human behavior and brain function (Burmester, Higgs, & Terry, 2018; Quintana, Smerud, Andreassen, & Djupesland, 2018). The majority of current research on IN-OT brain modulation, however, considers activity in isolated regions during a task (Koch, van Zuiden, et al., 2016; Riem et al., 2011; Zhao et al., 2016). Little is known about IN-OT effects on resting-state functional connectivity (rs-FC), i.e., temporal correlations in spontaneous fluctuations of blood oxygen level-dependent (BOLD) signals at rest, providing valuable insights into the inherent functional organization of the brain (Cabral et al., 2011). Further, the few existing IN-OT functional connectivity studies are mostly limited to coupling between two regions (amygdala and insula, De Cagna et al., 2019, nasal administration of 24 International Units (IUs), amygdala and medial prefrontal cortex, Ebner et al., 2016, nasal administration of 24 IUs; thalamus and amygdala, Koch et al., 2019, nasal administration of 40 IUs; but see Bethlehem et al., 2017, nasal administration of 24 IUs; Brodmann et al., 2017, nasal administration of 24 IUs).

Complex brain function, however, is subserved by large-scale brain networks (Bellec et al., 2006), such as the salience network (Menon, 2015), the default mode network (Andrews-Hanna et al., 2014), and the basal ganglia–thalamus network (Haber & Calzavara, 2009; Luo et al., 2012). These networks support social function (Di Simplicio et al., 2009; Grimm et al., 2009; Ince et al., 2023; Kirkpatrick et al., 2014), rendering them particularly likely and effective targets of IN-OT modulation. However, a large-scale network approach (both regarding within as well as between network coupling) to the study of IN-OT brain modulation is nascent (for a recent summary see Liu, Lin, Feifel, & Ebner, 2022, nasal administration of 24 IUs) and IN-OT’s wider-ranging brain mechanisms of action are still insufficiently understood.

Furthermore, previous IN-OT functional connectivity studies exclusively focused on young and middle-aged adults, despite increasing evidence of age-differential effects of IN-OT on both human brain and behavior (Ebner, Maura, MacDonald, Westberg, & Fischer, 2013; Horta, Pehlivanoglu, & Ebner, 2020; Sannino, Chini, & Grinevich, 2017). One exception is Liu et al. (2022) which comprised young and older participants and found that acute (i.e., single-dose) IN-OT decreased rs-FC of the right insula with the salience network in both age groups as well as of the left amygdala with the salience network in older adults. Acute IN-OT also decreased rs-FC between the angular gyrus and the default mode network in both young and older adults. Note that Liu et al. did not examine effects in the basal ganglia–thalamus network, despite evidence of enhanced rs-FC within this network after acute IN-OT in young adults (Bethlehem et al., 2017; Rocchetti et al., 2014).

Finally, only two studies to date have investigated effects of chronic (i.e., repeated) IN-OT on brain activity (Kou et al., 2022, nasal administration for 3 or 5 days with 24 IUs per day) and rs-FC (Watanabe et al., 2015, nasal administration for 6 weeks with 24 IUs twice a day for a total of 48 IUs per day) in humans, and none in aging. To probe IN-OT’s treatment potential, however, determination of chronic IN-OT effects on brain functional connectivity, including among older adults, is warranted (Horta et al., 2020 1).

To fill these research gaps, the present study examined effects of a four-week chronic IN-OT on both within- and between-network rs-FC of the salience network, the default mode network, and the basal ganglia–thalamus network in a sample of generally healthy older adults. Building on Liu et al. (2022), we also directly compared effects from chronic relative to acute IN-OT on within- and between-network connectivity in older adults; as chronic administration of any bioactive drug may produce either more pronounced or less pronounced (tolerance) effects compared to acute administration (Brusa et al., 2007). Chronic IN-OT’s effects may be pronounced over acute effects due to Chronic IN-OT’s potential for prolonged exposure to the OT (Peters et al., 2014), increased receptor sensitivity (Zanos et al., 2014), and/or neuroplastic changes linked to a decrease in epigenetic methylation of the OT receptor and an increase in OT receptor expression (Alaerts et al., 2023, nasal administration for 28 days with 12 IUs twice a day for a total of 24 IUs per day).

We expected chronic IN-OT to modulate within- (Hypothesis 1a) and between- (Hypothesis 1b) network rs-FC for all three networks (i.e., salience network, default mode network, and basal ganglia–thalamus network). We further hypothesized more pronounced effects of chronic than acute IN-OT effects on both within- (Hypothesis 2a) and between- (Hypothesis 2b) network rs-FC for all three networks. Beyond delineation of the effects of chronic IN-OT on a single specific brain network (within-network) or interaction between two brain networks (between-network), we also explored chronic IN-OT effects on resting-state functional coupling between all three networks using selective rs-FC, a comparatively novel metric referring to specific brain networks displaying stronger connections with distinct networks compared to others, indicative of more specialized, efficient network communication (Simmons et al., 2013; Chan et al., 2014). We did not formulate hypotheses for this outcome measure, however, given the lack of prior literature on this variable in OT and aging.

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