Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Wednesday, August 21, 2024

Anti-inflammatory diets could lower dementia risk among older individuals with cardiometabolic disorders

 Useless, NOTHING SPECIFIC IN HERE!

If you want to be useful you create an EXACT diet protocol, this does nothing towards that.  

Your chances of getting dementia. YOUR DOCTOR IS RESPONSIBLE FOR PREVENTING THIS!

1. A documented 33% dementia chance post-stroke from an Australian study?   May 2012.

2. Then this study came out and seems to have a range from 17-66%. December 2013.`    

3. A 20% chance in this research.   July 2013.

4. Dementia Risk Doubled in Patients Following Stroke September 2018 

The latest here:

Anti-inflammatory diets could lower dementia risk among older individuals with cardiometabolic disorders

In a recent study published in JAMA Network Open, researchers investigated whether anti-inflammatory diets support cognitive functions among cardiometabolic disease (CMD) patients.

Study: Anti-Inflammatory Diet and Dementia in Older Adults With Cardiometabolic Diseases. Image Credit: Elena Eryomenko/Shutterstock.comStudy: Anti-Inflammatory Diet and Dementia in Older Adults With Cardiometabolic Diseases. Image Credit: Elena Eryomenko/Shutterstock.com

Background

CMDs like insulin-independent diabetes, heart disease, and stroke are associated with an elevated dementia risk, particularly when they coexist. Studies associate inflammation with the pathophysiologic features of CMDs and dementia, and dietary habits can modulate systemic inflammation.

Increased levels of inflammatory biomarkers are associated with Western diets heavy on eggs, high-fat dairy, red meat, processed foods, and refined grains.

In contrast, diets rich in fruits, vegetables, whole grains, seafood, and legumes lower inflammatory biomarker levels.

Studies associate lower dietary inflammation with a lower risk of cognitive decline and favorable MRI brain aging indicators. However, the efficacy of anti-inflammatory diets in improving cognition in CMD patients is uncertain.

About the study

The present study researchers explored the potential cognitive benefits of anti-inflammatory diets among older adults with cardiometabolic diseases.

The researchers included 84,342 adult United Kingdom Biobank participants aged 60 years or above, with baseline evaluations conducted between 13 March 2006 and 1 October 2010.

They excluded individuals with dementia, insulin-dependent diabetes, and those with missing CMD data. The primary study outcome was new-onset dementia, identified through medical records and self-reported.

The researchers performed joint effects analysis to assess cardiometabolic disease status and diet-related inflammation concerning the risk of dementia and magnetic resonance imaging (MRI) markers of neurodegeneration and vascular injury in the brain.

In total, 8,917 participants without chronic neurological diseases underwent MRI between 2 May 2014 and 13 March 2020 to provide their gray matter volumes (GMV), total brain volume (TBV), hippocampal volumes (HV), white matter volumes (WMV), and white matter hyperintensity volumes (WMHV)

Researchers ascertained baseline CMDs such as heart disease, stroke, and insulin-independent diabetes from health records.

They calculated Dietary Inflammatory Index (DII) scores based on 31 food nutrients consumed between 8 February 2011 and 15 June 2012, evaluated using the 24-hour Oxford WebQ dietary assessment.

Points up to -1.5 indicated anti-inflammatory, above -1.5 but less than 0.5 indicated neutral, and 0.5 or above indicated pro-inflammatory potential.

Researchers followed the participants through 20 January 2022. They used Cox proportional hazard regressions to calculate the hazard ratios (HR) for analysis. Study covariates included race, education, body mass index (BMI), smoking status, calorie intake, physical activity, socioeconomic status, hypertension, antihypertensive medication use, and apolipoprotein E (APOE) status.

MRI covariates included time between scans, assessment centers, and table and head positions within MRI scanners.

Researchers performed sensitivity analyses applying Fine and Gray's regressions to non-imputed information and limiting participants to those completing at least two dietary evaluations, excluding individuals diagnosed with dementia within five years.

They also determined the association of DII scores with systemic inflammation biomarkers such as serological C-reactive protein (CRP) levels and assessed DII score stability across dietary evaluations.

Results

The mean participant age was 64 years; 51% were female, and 37% had attained at least a college-level education. At baseline, 14,079 (17%) had one or more cardiometabolic diseases.

CMD patients tended to be less educated, older, physically inactive, and more socioeconomically deprived male Asian black smokers with elevated BMI and hypertension. Individuals undergoing MRI had lower age, less socioeconomic deprivation, and better vascular injury risk profiles.

Over 12 years (median) of follow-up, 1,559 individuals (1.90%) received dementia diagnosis. The joint effects analysis yielded an HR of 2.4 for dementia for CMD patients following pro-inflammatory diets and 1.7 for individuals with CMDs consuming foods with anti-inflammatory potential.

Individuals with CMDs consuming anti-inflammatory diets rather than pro-inflammatory diets showed a 31% lower dementia risk (HR, 0.7) among CMD patients consuming anti-inflammatory foods.

Brain MRI scans of individuals consuming anti-inflammatory diets showed significantly higher GMV and lower WMHV values. Using Laplace regressions, CMD patients eating anti-inflammatory foods developed dementia two years after those with CMDs who ate pro-inflammatory foods.

Sensitivity analyses yielded similar results with stable DII scores across dietary evaluations. However, Fine and Gray's regressions yielded attenuated all-cause dementia HR values, indicating that the competing risk of death could influence the results.

Conclusions

The study found that cardiometabolic disease patients consuming anti-inflammatory foods had a decreased incidence of dementia, higher GMV, and lower WMHV values, indicating less neurodegeneration and vascular damage.

The findings suggest that anti-inflammatory diets improve cognitive function in those with cardiometabolic illnesses. Anti-inflammatory foods may reduce systemic inflammation, delaying dementia onset.

Longitudinal brain MRI studies may enhance knowledge of the links between dietary inflammation and brain illness, particularly concerning risk factors like cardiometabolic disorders.

Journal reference:

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