Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Saturday, August 24, 2024

Brain Thinning Predicts Alzheimer’s 10 Years Before Symptoms

 

With your chances of getting dementia post stroke. YOUR DOCTOR IS RESPONSIBLE FOR PREVENTING THIS!

And they should be measuring your cortical thickness to see the protocols that are needed to restore back to normal thickness! If your doctor doesn't have those protocols; you don't have a functioning stroke doctor!

1. A documented 33% dementia chance post-stroke from an Australian study?   May 2012.

2. Then this study came out and seems to have a range from 17-66%. December 2013.`    

3. A 20% chance in this research.   July 2013.

4. Dementia Risk Doubled in Patients Following Stroke September 2018 

The latest here:

Brain Thinning Predicts Alzheimer’s 10 Years Before Symptoms

Summary: Researchers identified cortical gray matter thinning as a potential early biomarker for dementia. In a study involving 1,500 participants from diverse backgrounds, thinner cortical gray matter was linked to a higher risk of developing dementia 5 to 10 years before symptoms appeared.

This finding suggests that measuring gray matter thickness via MRI could be key in early dementia detection and intervention. The research highlights the importance of early diagnosis in managing and possibly slowing the progression of dementia.

Key Facts:

  1. Cortical gray matter thinning is a promising biomarker for identifying individuals at high risk of dementia 5 to 10 years before symptoms manifest.
  2. The study’s findings were consistent across diverse racial and ethnic groups, enhancing the biomarker’s potential applicability.
  3. This discovery opens new avenues for early intervention, lifestyle modifications, and the development of targeted therapeutics for dementia.

Source: UT San Antonio

A ribbon of brain tissue called cortical gray matter grows thinner in people who go on to develop dementia, and this appears to be an accurate biomarker of the disease five to 10 years before symptoms appear, researchers from The University of Texas Health Science Center at San Antonio (also called UT Health San Antonio) reported.

The researchers, working with colleagues from The University of California, Davis, and Boston University, conducted an MRI brain imaging study published in Alzheimer’s & Dementia: The Journal of the Alzheimer’s Association. 

They studied 1,000 Massachusetts participants in the Framingham Heart Study and 500 people from a California cohort. The California volunteers included 44% representation of Black and Hispanic participants, whereas the Massachusetts cohort was predominantly non-Hispanic white. Both cohorts were 70 to 74 years of age on average at the time of MRI studies.

This shows an older man.
Repeating the Framingham findings in the more-diverse California cohort “gives us confidence that our results are robust,” Satizabal said. Credit: Neuroscience News

“The big interest in this paper is that, if we can replicate it in additional samples, cortical gray matter thickness will be a marker we can use to identify people at high risk of dementia,” said study lead author Claudia Satizabal, PhD, of UT Health San Antonio’s Glenn Biggs Institute for Alzheimer’s and Neurodegenerative Diseases.

“By detecting the disease early, we are in a better time window for therapeutic interventions and lifestyle modifications, and to do better tracking of brain health to decrease individuals’ progression to dementia.”

Repeating the Framingham findings in the more-diverse California cohort “gives us confidence that our results are robust,” Satizabal said.

Sifting MRIs for a pattern

While dementias can affect different brain regions, Alzheimer’s disease and frontotemporal dementia impact the cortex, and Alzheimer’s is the most common type of dementia.

The study compared participants with and without dementia at the time of MRI. “We went back and examined the brain MRIs done 10 years earlier, and then we mixed them up to see if we could discern a pattern that reliably distinguished those who later developed dementia from those who did not,” said co-author Sudha Seshadri, MD, director of the Glenn Biggs Institute at UT Health San Antonio and senior investigator with the Framingham Heart Study.

“This kind of study is only possible when you have longitudinal follow-up over many years as we did at Framingham and as we are building in San Antonio,” Seshadri said. “The people who had the research MRI scans while they were well and kept coming back to be studied are the selfless heroes who make such valuable discoveries, such prediction tools possible.”

The results were consistent across populations. Thicker ribbons correlated with better outcomes and thinner ribbons with worse, in general. “Although more studies are needed to validate this biomarker, we’re off to a good start,” Satizabal said. “The relationship between thinning and dementia risk behaved the same way in different races and ethnic groups.”

Applications

Clinical trial researchers could use the thinning biomarker to minimize cost by selecting participants who haven’t yet developed any disease but are on track for it, Seshadri said. They would be at greatest need to try investigational medications, she said.

The biomarker would also be useful to develop and evaluate therapeutics, Seshadri noted.

Future directions

Satizabal said the team plans to explore risk factors that may be related to the thinning. These include cardiovascular risk factors, diet, genetics and exposure to environmental pollutants, she said.

“We looked at APOE4, which is a main genetic factor related to dementia, and it was not related to gray matter thickness at all,” Satizabal said. “We think this is good, because if thickness is not genetically determined, then there are modifiable factors such as diet and exercise that can influence it.”

Derived in clinical MRIs

Could the MRI gray matter biomarker be used widely someday?

“A high proportion of people going to the neurologist get their MRI done, so this thickness value might be something that a neuroradiologist derives,” Seshadri said. “A person’s gray matter thickness might be analyzed as a percentile of the thickness of healthy people for that age.”

Acknowledgments

National Institutes of Health/National Institute on Aging funding for Alzheimer’s Disease Research Centers (ADRCs) at The University of Texas Health Science Center at San Antonio; The University of California, Davis; and Boston University School of Medicine supported this study.

About this Alzheimer’s disease research news

Author: Steven Lee
Source: UT San Antonio
Contact: Steven Lee – UT San Antonio
Image: The image is credited to Neuroscience News

Original Research: Open access.
A novel neuroimaging signature for ADRD risk stratification in the community” by Claudia Satizabal et al. Alzheimer’s & Dementia

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