Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Friday, March 9, 2018

Modules and brain mapping

A more understandable writeup on the research; Your doctor can figure out how to apply this to your recovery.

What Does Any Part of the Brain Do?

The actual research;

Modules and brain mapping

Abstract

This review highlights the key role of modularity and the additive factors method in functional neuroimaging. Our focus is on structure–function mappings in the human brain and how these are disclosed by brain mapping. We describe how modularity of processing (and possibly processes) was a key point of reference for establishing functional segregation as a principle of brain organization. Furthermore, modularity plays a crucial role when trying to characterize distributed brain responses in terms of functional integration or coupling among brain areas. We consider additive factors logic and how it helped to shape the design and interpretation of studies at the inception of brain mapping, with a special focus on factorial designs. We look at factorial designs in activation experiments and in the context of lesion–deficit mapping. In both cases, the presence or absence of interactions among various experimental factors has proven essential in understanding the context-sensitive nature of distributed but modular processing and discerning the nature of (potentially degenerate) structure–function relationships in cognitive neuroscience.
Keywords: Additive factors, Modularity, Factorial, Connectivity, Degeneracy
This review is essentially a narrative about how some of the fundaments of experimental design and interpretation of human brain mapping studies have developed over the past two decades. Its focus is on the role of modularity and additive factors logic in guiding these developments. This is a somewhat self-referential account, which allows us to describe how our earlier misconceptions gave way to more enduring perspectives— perspectives that help guide our current research into structure–function relationships in the brain.
This review comprises four sections. The first considers, briefly, the rationale for modularity and its place within distributed neuronal processing architectures. We consider evolutionary imperatives for modularity and then a slightly more abstract treatment that underpins the analysis of functional and effective connectivity. The second section is a short historical perspective that covers the rise and fall of cognitive subtraction and the emergence of factorial designs in neuroimaging. Our focus here is on the role of additive factors logic and the connection to conjunction analyses in neuroimaging. The third section pursues the importance of interactions in factorial designs—specifically, their role in disclosing context-sensitive interactions or coupling among modular brain areas. We illustrate this using the notion of dynamic diaschisis and psychophysiolopical interactions. The final section turns to lesion–deficit mapping and neuropsychology (in the sense of using lesions to infer functional architectures). Here, we review the concept of necessary and sufficient brain systems for a given task and how these led to the appreciation of degenerate structure–function mappings. Additive factors logic again plays a key role but, in this instance, the combination rule (Sternberg, 2011 this issue) becomes probabilistic and acquires a multiplicative aspect. We rehearse the importance of degenerate mappings in the context of multilesion–deficit analysis and conclude with some comments on the role of cognitive ontologies in making the most of neuroimaging data.

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