Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Wednesday, June 11, 2014

Air pollution not linked to STEMI, stroke

I bet I could prove breathing is tied to stroke.
Earlier reports here;
Air Pollution Tied to Stroke, Cognitive Slide
 And debunked here: Although they don't say why it is bad
More bad statistics presented as science by a gullible media.
http://junkscience.com/2012/02/14/warning-over-air-pollution-link-to-stroke-research/

The latest pronouncements here:
http://www.clinicaladvisor.com/air-pollution-not-linked-to-stemi-stroke/article/351491/?
Ambient air pollution has short-term effects on specific cardiovascular disease (CVD) events, but not others, according to a study published in Heart.
Ai Milojevic, PhD, from the London School of Hygiene & Tropical Medicine, and colleagues examined the short-term correlation between ambient air pollution and a range of cardiovascular events. Data were collected from three national databases in England and Wales.
More than 400,000 myocardial infarction (MI) events from the Myocardial Ischaemia National Audit Project (MINAP) database, over two million CVD emergency hospital admissions, and more than 600,000 CVD deaths were linked to daily air pollutant data.
With respect to mortality outcomes, no CVD outcome analyzed correlated clearly with any pollutant, with the exception of particulate matter less than 2.5 µm in aerodynamic diameter, which correlated with arrhythmias, atrial fibrillation, and pulmonary embolism, the researchers found.
Only nitrogen dioxide (NO2) correlated with an increased risk of hospital admission, with a 10th to 90th centile increase correlating with increased risk for CVD (1.7%; 95% CI: 0.9-2.6), non-MI CVD (2%; 95% CI:1.1-2.9), arrhythmias (2.9%;  95% CI: 0.6-5.2), atrial fibrillation (2.8%; 95% CI: 0.3 to 5.4), and heart failure (4.4%; 95% CI:2.0 to 6.8).
An increased risk of MI was seen only for NO2, which was specific for non-ST-segment elevation myocardial infarction (3.6%; 95% CI: 0.4 to 6.9]).
"The strongest associations with air pollution were observed with selected non-MI outcomes," the researchers wrote.
In an accompanying editorial, Anoop Shah, MD, and David E. Newby, MD, of the Royal Infirmary of Edinburgh in the United Kingdom, summarized three important take-home points from the latest study data:
  • There is no clear evidence that short-term increases in air pollutants influenced overall CV or HF
  • There is no clear association between ambient air pollutants and death or hospital admission with STEMI, stroke or HF
  • The overall association between air pollutants and CV morbidity and mortality remained unchanged
"The current lack of consistent associations with contemporary UK data may suggest that as the fog begins to clear, the adverse health effects of air pollution are starting to have less of an impact and are more difficult to delineate," Shah and Newby wrote.

References


  • Milojevic S et al. Heart. doi:10.1136/heartjnl-2013-304963.
  • Shah A, Newby DE. Heart. doi:10.1136/heartjnl-2014-305877

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