Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Thursday, August 21, 2014

How Stress Promotes Atherosclerosis

You will need to demand that your doctor remove all the stressors from your hospital environment.
1. Poor sleep
2. No objective diagnosis of damage.
3. No path to 100% recovery.
4. Nocebo comments about no further recovery/plateau.
5. Worries about cognitive decline with no stroke protocol to fix that.
6. Fatigue.
7. Spasticity which your doctor has no idea how to cure.

http://www.united-academics.org/magazine/health-medicine/how-stress-promotes-atherosclerosis/
Atherosclerosis is an inflammatory disease characterized by the “hardening” of the arteries. This is caused by the accumulation of deposits mainly composed of cell debris, fats, cholesterol, calcium, on the inner walls of arteries leading to vessels’ narrowing and decreased blood flow. These deposits are called plaques and they also contain platelets and immune cells (leukocytes), in particular inflammatory monocytes and macrophages. Enzymes produced by the immune cells can partially degrade the plaques causing their disruption and cause formation of blood clots that will occlude the vessels and block oxygen supply to different organs and tissues. If a clot occurs in vessels of the heart or the brain, it will elicit a heart attack or a stroke respectively.
Link between stress and atherosclerosis
There is evidence that chronic stress increases the risk of atherosclerosis, but no mechanism linking the two phenomena has been demonstrated so far. Since stressful emotional states can affect the function of the immune system, Heidt and colleagues of the Massachusetts General Hospital hypothesized that stress increases the activity of inflammatory cells in the plaques facilitating their rupture, as you can read in their recently published article.
To test this possibility the scientists measured the effects of various forms of stress (including isolation, damp bedding, overnight illumination) on the number of leukocytes in mice. They found that compared to the controls the stressed animals had significantly more circulating immune cells. This was the result of increased division of the hematopoietic stem cells (HSCs), which are the immature precursors of the immune cells, and subsequent increased production of leukocyte progenitors in their bone marrow.

More at link.



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