Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Saturday, June 16, 2018

Study finds mixed associations between alcohol intake, cardiovascular events

Your doctor can explain this all to you.

Study finds mixed associations between alcohol intake, cardiovascular events

New research shows an inverse link between alcohol intake and risk of coronary heart disease (CHD), but a positive association with stroke risk.
"Given the known positive association of alcohol intake with all cause mortality and the risk of cancer, the opposing associations of alcohol intake we found with different cardiovascular disease types strengthen the rationale for policies to reduce alcohol consumption," researchers conclude in a report online May 29 in The BMJ.
Dr. Pietro Ferrari from the International Agency for Research on Cancer (IARC) and colleagues investigated associations between drinking alcohol and risk of incident non-fatal and fatal CHD and stroke in more than 32,500 adults (ages 35-70; 52% women) without cardiovascular disease (CVD) at baseline in eight European countries in the EPIC-CVD study.
Among the total cohort, there were 9,307 non-fatal CHD events, 1,699 fatal CHD events, 5,855 non-fatal strokes, and 733 fatal strokes.
Alcohol intake at baseline was inversely associated with nonfatal CHD (HR, 0.94 per 12 g/day higher intake; 95% CI, 0.92 to 0.96).
For fatal CHD, there was a J-shaped relationship between baseline alcohol intake and risk. The hazard ratio was 0.83 (95% CI, 0.70 to 0.98) for 5.0 to 14.9 g/day, 0.65 (95% CI, 0.53 to 0.81) for 15.0 to 29.9 g/day, and 0.82 (95% CI, 0.65 to 1.03) for 30.0 to 59.9 g/day of total alcohol intake, compared with intake of 0.1 to 4.9 g/ day.
In contrast, hazard ratios for non-fatal and fatal stroke were 1.04 (95% CI, 1.02 to 1.07) and 1.05 (95% CI, 0.98 to 1.13), respectively, per 12 g/day increase in baseline alcohol intake, with results similar for ischemic and hemorrhagic stroke.
The associations with cardiovascular outcomes were also broadly similar with average lifetime alcohol intake as for baseline intake, and across the eight countries studied, the researchers say.
This study, they conclude, highlights the "opposing associations" of alcohol intake with different types of cardiovascular disease and "strengthens the evidence for policies to reduce alcohol consumption."
The study had no commercial funding and the authors have no conflicts of interest.

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