Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Thursday, July 15, 2021

Dementia Comes 5 Years Later for Some

With your risk of dementia post stroke your doctor should have  a protocol on this. If not they need to be fired.

Your risk of dementia, has your doctor told you of this?

1. A documented 33% dementia chance post-stroke from an Australian study?   May 2012.

2. Then this study came out and seems to have a range from 17-66%. December 2013.`    

3. A 20% chance in this research.   July 2013.

4. Dementia Risk Doubled in Patients Following Stroke September 2018 

The latest here:

Dementia Comes 5 Years Later for Some

Reading, processing information in old age may have benefit

A senior woman reading a book and drinking a mug of tea.

A cognitively active lifestyle that involves reading and processing information in old age may delay the onset of dementia in Alzheimer's disease by as much as 5 years, a longitudinal study suggested.

Older adults who had the highest level of late-life cognitive activity had a mean onset age of Alzheimer's dementia of 94, reported Robert Wilson, PhD, of Rush University Medical Center in Chicago, and colleagues.

In contrast, those with the lowest late-life cognitive activity levels developed dementia at age 89, they wrote in Neurology.

"Our study shows that people who engage in more cognitively stimulating activities may be delaying the age at which they develop dementia," Wilson said in a statement.

"It is important to note, after we accounted for late-life level of cognitive activity, neither education nor early-life cognitive activity were associated with the age at which a person developed Alzheimer's dementia," he continued. "Our research suggests that the link between cognitive activity and the age at which a person developed dementia is mainly driven by the activities you do later in life."

"This study provides further support for the concept of cognitive reserve, where genetic and life exposures allow some people to cope better than others with age- or disease-related brain changes," noted Yaakov Stern, PhD, of Columbia University in New York City, who wasn't involved with the research.

"Previous work has suggested many exposures that can contribute to cognitive reserve, including education, aspects of occupational attainment, and late-life leisure activities, along with IQ," Stern told MedPage Today.

"What is interesting here is that this paper suggests that late-life cognitive activity can contribute to this reserve," he added. "Further, their autopsy data indicates that the late-life cognitive activity does not influence the pathology itself, making it more likely that it influences cognitive reserve -- the ability to cope with the pathology."

The study involved 1,903 older adults from the longitudinal Rush Memory and Aging Project who were dementia-free at enrollment. Participants had annual clinical evaluations to diagnose dementia and Alzheimer's disease. Those who died had a neuropathologic examination.

Mean age at baseline was 79.7. Participants had completed an average of 15 years of formal education; most were women (74.9%) and white (89.1%). They had a median of one of seven chronic medical conditions such as cancer or heart disease, and a median income between $35,000 and $49,999.

At baseline, participants reported how frequently they participated in seven specific cognitively stimulating activities on a 5-point scale. The activities emphasized seeking or processing information: daily reading, or time spent each year visiting a library, reading newspapers, reading magazines, reading books, writing letters, or playing games like puzzles, cards, and board games. Scores ranged from 1 (no time) to 5 (every day or almost every day).

People in the 90th percentile of cognitive activity scored an average of 4.0, indicating they were involved with these activities several times per week. Those with the lowest cognitive activity -- the 10th percentile -- had an average score of 2.1, indicating activities several times per year.

Over a mean of 6.8 years of annual followup evaluations, 457 people were diagnosed with incident Alzheimer's dementia at a mean age of 88.6 (range 64.1-106.5). In an extended accelerated failure time model, a higher level of baseline cognitive activity (mean 3.2) was associated with older age at Alzheimer's dementia onset (estimate 0.026, 95% CI 0.013-0.039).

An activity score of 2.1 (10th percentile) was linked with an average dementia onset age of 88.6. A activity score of 4.0 (90th percentile) was associated with an average onset age of 93.6. Cognitive activity continued to be related to age of Alzheimer's dementia onset after adjusting for sex, education, APOE4 status, and baseline social activity and loneliness. The association persisted even when people who had mild cognitive impairment at baseline were excluded.

In the 695 participants who died and had a neuropathologic examination, cognitive activity was unrelated to postmortem markers of Alzheimer's and other dementias, including amyloid burden and tau.

Overall, the bases of observed associations between cognitive activity and age of Alzheimer's dementia onset are uncertain, Wilson and colleagues noted.

"One possibility is that low cognitive activity is an early sign of Alzheimer's disease (reverse causality hypothesis)," they wrote. "In the present analyses, however, postmortem markers of Alzheimer's disease and other dementias were unrelated to the self-report measure of cognitive activity, consistent with prior research in this cohort."

"We think a more likely possibility is that cognitive activities lead to changes in brain structure and function that enhance cognitive reserve," the researchers added.

The main limitation of the study is that analyses are based on a selected group of mainly white, well-educated participants. "Further research will be needed to establish whether the findings will generalize to more diverse cohorts with a wider range of age and cognitive experiences," they wrote.

  • Judy George covers neurology and neuroscience news for MedPage Today, writing about brain aging, Alzheimer’s, dementia, MS, rare diseases, epilepsy, autism, headache, stroke, Parkinson’s, ALS, concussion, CTE, sleep, pain, and more. Follow

Disclosures

This study was supported by grants from the National Institute on Aging.

The researchers had no disclosures to report.

 

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