With your 33% dementia chance post-stroke from an Australian study
your doctor, if any good at all, should find out your baseline immediately post-stroke and then evaluate your changes over the years. Prevention and delaying that dementia is your doctors responsibility. However they will not accept that responsibility, so you are on your own. My ideas on that are here;
Dementia prevention 19 ways
The diagnostic tool here;
http://www.alphagalileo.org/ViewItem.aspx?ItemId=142491&CultureCode=en
A new diagnostic tool helps clinicians to differentiate between
Alzheimer's disease, frontotemporal dementia and mild cognitive
impairment. Presented in the doctoral thesis of MD Miguel Ángel Muñoz
Ruiz at the University of Eastern Finland, the new method consists of a
Disease State Index combining data from multiple sources, and of a
Disease State Fingerprint showing the findings in a visual format.
It is estimated that more than 35.6 million people were living with
dementia worldwide in 2010. This number will increase in the coming
years and there is a need to identify these patients to provide them
with proper treatment and care from the very beginning of the disease.
The differential diagnosis of the dementia diseases represents a
challenge particularly in the early phases. Many studies have focused on
predicting the possible conversion from mild cognitive impairment, a
pre-dementia stage, to Alzheimer's disease (AD), the most common
dementia disease. Several methods have also been proposed for
differentiating between AD and frontotemporal dementia (FTD), another
relatively common degenerative dementia. An early and precise diagnosis
of these two dementia diseases is needed in order to benefit from
treatments designed to influence the disease mechanisms.
In the recent years, important advances have been made especially in
the development of new diagnostic methods. Several biomarkers and tests
are used in the clinical practice, such as cerebrospinal fluid
biomarkers, imaging methods, genetic profiling and neuropsychological
tests. However, making a differential diagnosis is not easy due to
overlapping clinical and biomarker findings and the unavoidable
subjective component when a clinician interprets all this multitude of
data. Furthermore, there is no single biomarker or test which could
clearly define whether a patient is suffering from AD or FTD.
The thesis of Dr Muñoz Ruiz introduces a new combination of different
methods for the differential diagnosis of AD, mild cognitive impairment
and FTD, and describes a tool comprising a Disease State Index and its
visual counterpart, a Disease State Fingerprint.
The Disease State Index encompasses all the data from multiple
sources while taking into account the most relevant method or test, and
the Disease State Fingerprint shows the findings in an easy-to-interpret
visual format.
The software combines data from multiple sources such as
psychological tests and brain MRI, and uses this data to create a
Disease State Index. The index is a numerical value between 0 and 1. In a
healthy person, the index is close to 0, while an index close to 1 is
an indicator of a dementia disease. The Disease State Fingerprint shows
the findings in an easy-to-interpret format in which the key findings
are clearly indicated by colour and size.
With the help of the new diagnostic tool, clinicians could know which
methods are more relevant for profiling a patient with a certain
dementia disease, i.e. whether it is mild cognitive impairment, FTD or
AD, and already at the first visit, the clinician could make a first
diagnosis for starting treatment and giving counselling to the patient.
The
original articles were published in PloS ONE and Journal of Alzheimer's
Disease. Two submitted articles were also presented.
Full bibliographic informationStructural
MRI in Frontotemporal Dementia: Comparisons between Hippocampal
volumetry, Tensor-based morphometry and Voxel-based morphometry. PLoS
ONE 7(12): e52531.
Disease Fingerprint in frontotemporal
degeneration with reference to Alzheimer's disease and mild cognitive
impairment. J Alzheimers Dis. 2013 Jan 1;35(4):727-39. doi:
10.3233/JAD-122260.
Use the labels in the right column to find what you want. Or you can go thru them one by one, there are only 29,112 posts. Searching is done in the search box in upper left corner. I blog on anything to do with stroke.DO NOT DO ANYTHING SUGGESTED HERE AS I AM NOT MEDICALLY TRAINED, YOUR DOCTOR IS, LISTEN TO THEM. BUT I BET THEY DON'T KNOW HOW TO GET YOU 100% RECOVERED. I DON'T EITHER, BUT HAVE PLENTY OF QUESTIONS FOR YOUR DOCTOR TO ANSWER.
Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.
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