Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Saturday, April 23, 2016

Transfer of gut bacteria affects brain function and nerve fiber insulation

So maybe you want a fecal transplant from a happy person to treat your depression. I'm assuming that this would work the same way in reverse as this research shows.
http://www.mdlinx.com/internal-medicine/medical-news-article/2016/04/22/6639469/?news_id=2386&

The Mount Sinai Hospital, 04/22/2016
Specific combinations of gut bacteria produce substances that affect myelin content and cause social avoidance behaviors in mice, according to a study conducted at the Icahn School of Medicine at Mount Sinai and published in the journal eLife. This research suggests that targeting intestinal bacteria, or their metabolites, could be one way to treat debilitating psychiatric disorders and demyelinating diseases, like multiple sclerosis. This current study led by Patrizia Casaccia, MD, PhD, Professor of Neuroscience, Genetics and Genomics, and Neurology, and Chief of the Center of Excellence for Myelin Repair, and post–doctoral fellow Mar Gacias, PhD, identifies bacteria–derived gut metabolites that can affect myelin content in the brains of mice and induce depression–like symptoms. Researchers transferred fecal bacteria from the gut of depressed mice to genetically distinct mice exhibiting non–depressed behavior. The study showed that the transfer of microbiota was sufficient to induce social withdrawal behaviors and change the expression of myelin genes and myelin content in the brains of the recipient mice. In an effort to define the mechanism of gut–brain communication, researchers identified bacterial communities associated with increased levels of cresol, a substance that has the ability to pass the blood–brain barrier. When the precursors of myelin–forming cells were cultured in a dish and exposed to cresol, they lost their ability to form myelin, thereby suggesting that a gut–derived metabolite impacted myelin formation in the brain.

No comments:

Post a Comment