Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Saturday, April 27, 2019

ESJ Comment: Therapeutic hypothermia for acute ischaemic stroke. Results of a European multicenter, randomized, phase III clinical trial

All my previous research posts on this suggested no useful intervention. Obviously no protocols were ever written on hypothermia so everyone is still shooting in the dark. The result being that survivors are still screwed with no consequences to the doctors who haven't written up protocols on this. Don't you just love incompetence?

 

ESJ Comment: Therapeutic hypothermia for acute ischaemic stroke. Results of a European multicenter, randomized, phase III clinical trial

Comment by Nicolas Martinez-Majander, Department of Neurology, Helsinki University Hospital, Finland
Original Article: H Bart van der Worp, Malcolm R Macleod, Philip MW Bath et al, 2019. Therapeutic hypothermia for acute ischaemic stroke. Results of a European multicenter, randomized, phase III clinical trial https://journals.sagepub.com/doi/10.1177/2396987319844690
Previous systematic reviews and meta-analyses of animal studies have shown that therapeutic hypothermia is highly effective in reducing infarct size and improving neurological outcome. Hypothermia can affect several ongoing processes in the penumbra, such as counteracting edema, reducing lactacidosis, and inhibiting free radical formation and apoptosis. However, there is only little evidence of therapeutic hypothermia in human stroke.
In this paper of ESJ, van der Worp and colleagues reported results of EuroHYP-1, a European multicentre, randomized, phase III clinical trial which aimed to assess whether modest systemic cooling started within 6 hours of symptom onset could improve functional outcome at three months in awake patients with acute ischaemic stroke. These patients were allocated to hypothermia (target body temperature of 34-35°C either with intravenous infusion or a pre-specified surface cooling method) within 6 h after onset of stroke. Hypothermia was maintained for 12 to 24 hours. The primary outcome was mRS score at 91 days, assessed by independent blinded adjudicators. Although the target sample size was 1500 patients, the trial was stopped after inclusion of 98 patients, at 23 study sites, because of slow recruitment. Of these, 49 were randomized to hypothermia and 49 to control arm. The intention-to-treat analysis showed no difference between the groups (OR for good outcome, 1.01; 95% CI, 0.48-2.13; p=0.97). 38% in the hypothermia group and 29% controls had at least one serious adverse event, such as pneumonia and symptomatic intracranial haemorrhage. Unfortunately however, the final sample was underpowered to detect any benefit or harm of therapeutic hypothermia. Furthermore, in about two thirds of patients randomized to hypothermia, it was not possible to achieve cooling target of body temperature of 34-35 °C, mainly because of shivering and discomfort. As the authors conclude, despite of a well-balanced and robust study protocol, the feasibility of cooling needs to be improved before launching any new trials.
Reference
H Bart van der Worp, Malcolm R Macleod, Philip MW Bath et al, 2019. Therapeutic hypothermia for acute ischaemic stroke. Results of a European multicenter, randomized, phase III clinical trial. European Stroke Journal. DOI: 10.1177/2396987319844690 https://journals.sagepub.com/doi/10.1177/2396987319844690

No comments:

Post a Comment