Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Tuesday, December 6, 2016

Study: Watch for PSE in Chronic Stroke Patients - post-stroke epilepsy

I didn't have to worry about this problem. You will need to ask exactly what areas were damaged  so you can inform your doctor of this PSE possibility.

Study: Watch for PSE in Chronic Stroke Patients - post-stroke epilepsy


Physicians should be on the lookout for epilepsy after chronic stroke, particularly in younger stroke patients and those with more brain damage, researchers reported here.
In a retrospective U.K. study cohort, about 11% of stroke patients developed post-stroke epilepsy (PSE), according to Beate Diehl, MD, PhD, of University College London (UCL), and colleagues at the American Epilepsy Society meeting.
Those who developed PSE were younger (44 versus 56) and had more extensive brain damage on MRI scans, they added.
"Many physicians treating stroke patients don't realize that falls, episodes of confusion, and loss of consciousness may be signs of post-stroke epilepsy," Diehl said in a statement. "Post-stroke epileptic seizures can negatively affect stroke recovery and rehabilitation."
Epilepsy often goes undiagnosed in the elderly, the researchers said. The leading cause of epilepsy in these older patients is stroke, which accounts for as much as 45% of epilepsy in people over age 60.
Although some risk factors for PSE have been identified, the exact mechanisms by which stroke causes epilepsy aren't known, Diehl and colleagues said. So they looked at data from the Predicting Language Outcome and Recovery After Stroke (PLORAS) database of more than 1,000 stroke patients in the U.K.
About 450 of these patients had 1 mm 3-voxel T1-weighted anatomical whole-brain scans acquired with a 3T MRI scanner at the UCL Wellcome Trust Center for Neuroimaging. Patients also answered questions about having seizures.
Overall, the researchers found that of the 369 patients who had left hemisphere strokes, 42 of them (11.4%) developed PSE.
The figure was similar for the 81 who had right-hemisphere strokes: nine of these patients (11.1%) developed PSE.
Patients who developed PSE were significantly younger than those without PSE (44 versus 56, P<0.0001), and PSE patients had twice the extent of brain damage of those without epilepsy -- their lesions were significantly larger (148 cm3 versus 73 cm3, P<0.0001) -- suggesting that the frequency of epilepsy increases with the size of the lesion.
When comparing lesion locations between PSE and non-PSE patients with left hemisphere stroke, the researchers found a region that was damaged in 64% of those with PSE, but this area was also damaged in 17% of those without PSE.
The authors said the incidence of PSE after damage to this region was only 33%.
Among those who had left hemispheric stroke and developed PSE, the most commonly affected areas of the brain were the basal ganglia -- particularly the globus pallidus and caudate nucleus -- and most nuclei of the thalamus (the anterior and ventral nuclei, as well as posterior regions including the pulvinar). The authors noted that these are important areas for controlling movement, sensation, and consciousness.
Their findings are in line with recent reports of a higher percentage of stroke patients developing epilepsy, they said, and concluded that doctors treating stroke patients should keep in mind that PSE is common, so they should watch for risk factors, such as extent of brain damage and younger age.

The study was supported by Epilepsy Research UK and the National Institute for Health Research UCL Hospitals Biomedical Research Center.
Diehl and co-authors disclosed no relevant relationships with industry.

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