Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Tuesday, April 5, 2011

anti-depressants linked to heart disease-stroke

This is rather depressing since other studies suggest than anti-depressants help recovery of motor control.
http://oc1dean.blogspot.com/2011/03/neurotransmitter-reboxetine-could.html
Reboxetine is a drug marketed as an antidepressant for use in the treatment of clinical depression, panic disorder and ADD/ADHD, developed by Pharmacia (now Pfizer).
Antidepressants thicken the arteries, linking use to heart disease and stroke
The findings come from Emory University researchers who conducted a study of twins to find the possible link between antidepressant use and heart attack and stroke.
Scientists study twins because they have the same genes, making it easier to sort out other contributing health risk factors such as smoking, diet and other lifestyle contributors. For the current study, the authors noted antidepressants may increase the risk of cardiovascular events, independent of depression that can also increase the risk of heart attack and stroke.
Carotid artery thickness increased with antidepressant use
For the study, the scientists measured thickness of the lining, or intima, in the carotid arteries that supply blood flow to the brain, using ultrasound. Measuring intima-media thickness is a reliable way to
Normally, intima-media thickness (IMT) progresses at the rate of 10 microns per year. According to first author Amit Shah, MD, a cardiology fellow at Emory University School of Medicine, "In our study, users of antidepressants see an average 40 micron increase in IMT, so their carotid arteries are in effect four years older."
Among 59 sets of twins, with one taking antidepressants, the researchers saw more thickening of the carotid arteries in those taking SSRI's like fluoxetine (60 percent of those taking antidepressants) and those who took other types.
Dr. Shah says, "I think we have to keep an open mind about the effects of antidepressants on neurochemicals like serotonin in places outside the brain, such as the vasculature. The body often compensates over time for drugs' immediate effects.
Antidepressants have a clinical benefit that has been established, so nobody taking these medications should stop based only on these results. This isn't the kind of study where we can know cause and effect, let alone mechanism, and we need to see whether this holds up in other population groups."
The study found a link between antidepressants and risk of heart attack and stroke and will be presented Tuesday, April 5 at the American College of Cardiology meeting in New Orleans.
The reason for the link between heart attack and stroke and antidepressant use may be from serotonin changes, but the researchers say the action of the hormone on the blood vessels is complex. Serotonin can either relax or narrow the blood vessels, depending on whether they are healthy or damaged.
So which one should we care about more? Recovery or another stroke or heart attack?  I hope my doctor has better insight into this especially since so many stroke patients need to be on anti-depressants.

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