Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Monday, August 28, 2017

Translational Stroke Research Vision and Opportunities

Whatever this report is it should be publicly available so stroke survivors can comment on it and make it better. But that won't happen, our stroke medical professionals think they know what needs to be done for stroke. Which is basically nothing, keep the status quo. No sense in rocking the boat.
http://stroke.ahajournals.org/content/48/9/2632?etoc=
Francesca Bosetti, James I. Koenig, Cenk Ayata, Stephen A. Back, Kyra Becker, Joseph P. Broderick, S. Thomas Carmichael, Sunghee Cho, Marilyn J. Cipolla, Dale Corbett, Roderick A. Corriveau, Steven C. Cramer, Adam R. Ferguson, Seth P. Finklestein, Byron D. Ford, Karen L. Furie, Thomas M. Hemmen, Costantino Iadecola, Lyn B. Jakeman, Scott Janis, Edward C. Jauch, Karen C. Johnston, Patrick M. Kochanek, Harold Kohn, Eng H. Lo, Patrick D. Lyden, Carina Mallard, Louise D. McCullough, Linda M. McGavern, James F. Meschia, Claudia S. Moy, Miguel A. Perez-Pinzon, Ipolia Ramadan, Sean I. Savitz, Lee H. Schwamm, Gary K. Steinberg, Mary P. Stenzel-Poore, Michael Tymianski, Steven Warach, Lawrence R. Wechsler, John H. Zhang, Walter Koroshetz
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See related article, p 2341
Stroke risk and poststroke disability have steadily decreased in the United States over the past 2 decades because of improved prevention and access to reperfusion therapies for acute ischemic stroke, such as tPA (tissue-type plasminogen activator; alteplase) and endovascular thrombectomy. Despite the efficacy and safety of thrombolysis and thrombectomy, not all patients who receive the treatment improve to full, independent recovery, and most patients are ineligible for treatment. Additionally, there are no efficacious treatments to improve long-term outcomes for patients after the acute phase of ischemic stroke or to reduce brain injury induced by acute intracerebral hemorrhage. Therefore, development of new therapies for both acute and chronic stroke is sorely needed.
Stroke occurs because of a variety of vascular pathologies and injury mechanisms, some of which are difficult to model in animals. With the exception of reperfusion therapy, preclinical research end points do not generally reflect clinical outcomes. Pharmacodynamics, pharmacokinetics, and target engagement in the human brain need to be further developed and optimized for stroke interventions so that drug level in brain tissue, time to initiation, and duration of treatment can be accurately measured in clinical trials. Many variables, such as heterogeneity of vascular pathologies, patient demographics, and a host of comorbid conditions, as well as the lack of validated biomarkers to stratify patient populations, limit the ability of typical stroke clinical trials to detect a treatment effect.
To address these gaps, the National Institute of Neurological Disorders and Stroke organized and sponsored the workshop Translational Stroke Research: Vision and Opportunities, which was held in Bethesda, Maryland, on November 1 to 2, 2016. The workshop gathered over 180 registered participants from academia, industry, the Food and Drug Administration, and other public and private funding agencies. …
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