Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Wednesday, December 15, 2021

Evaluation of Central Fatigue in Post-stroke Rehabilitation: A Pilot Study

Well we've known of post stroke fatigue for years. Why are we evaluating it rather than curing that fatigue? Does anyone in the stroke world actually think with their two functioning neurons? 

At least half of all stroke survivors experience fatigue Or is it 70%?

Or is it 40%?

Evaluation of Central Fatigue in Post-stroke Rehabilitation: A Pilot Study

Publisher: IEEE

Abstract:
Central fatigue induced by excessive rehabilitation training would limit motor activity or even damage the post-stroke motor function recovery. However, the central fatigue progress during training is unclear and ignored in post-stroke rehabilitation. In this study, we tried to investigate the changes in central fatigue with fractal dimension (FD) of electromyography (EMG) at different peripheral fatigue levels based on the intracerebral haemorrhage (ICH) model. Ten Sprague-Dawley rats with ICH and EMG electrodes implantation were randomly distributed into two groups: the forced training (FOR) group with exhausted peripheral fatigue level (n=5) and fatigue-controlled (FAT) group (n=5) with peripheral fatigue constrained in moderate level. A higher central fatigue level was found in the FOR group (P<0.0001), and the central fatigue could be alleviated by peripheral fatigue-based modulation in the FAT group. The FAT group with less central fatigue achieved significantly better motor function recovery (P<0.0001), and it might be related to the recovery in the ability of motor unit recruitments.
 

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