Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Friday, September 22, 2023

Blood, CSF Marker Detects Parkinsonian Disorders

With your risk of Parkinsons post stroke a competent doctor would do this test on you to establish a baseline reading.

Parkinson’s Disease May Have Link to Stroke March 2017

Do you prefer your doctor and hospital incompetence NOT KNOWING OR NOT DOING anything on this?

 

Blood, CSF Marker Detects Parkinsonian Disorders

Enzyme also identified preclinical Lewy body disease

A computer rendering of Lewy bodies inside a nerve cell.

A novel blood and cerebrospinal fluid (CSF) measure showed promise as a biomarker for Parkinsonian disorders.

CSF levels of DOPA decarboxylase (DDC) -- an enzyme that converts levodopa into dopamine -- accurately identified patients with Lewy body disease with an area under the curve (AUC) of 0.89, reported Oskar Hansson, MD, PhD, of Lund University in Sweden, and co-authors in a Nature Agingopens in a new tab or window research letter.

In an independent replication cohort, plasma DDC levels identified both Lewy body disease and atypical Parkinsonian disorders (AUC 0.92).

"This study shows for the first time that DDC is elevated in both cerebrospinal fluid and blood in patients with Parkinsonian disorders, including Parkinson's disease, Lewy body dementia, progressive supranuclear palsy, and multiple system atrophy," Hansson told MedPage Today. "We even found that the levels were increased before symptom onset and could predict subsequent development of clinical disease."

DDC, also known as aromatic L-amino acid decarboxylase, identified preclinical Lewy body disease in unimpaired individuals who had a positive alpha-synuclein seed amplification assay (AUC 0.81), the researchers said. It predicted progression to clinical Lewy body disease over a 3-year period in preclinical cases (HR 3.7 per standard deviation change, 95% CI 1.1–12.7, P=0.035).

DDC levels were increased in atypical Parkinsonian disorders but not in other neurodegenerative disorders like Alzheimer's disease, frontotemporal dementia, or vascular dementia.

"Parkinsonian disorders are often difficult to diagnose accurately based on clinical assessments alone, especially during early disease stages," Hansson noted.

"PET imaging of the dopaminergic neurons is often helpful, but it's expensive and requires a complex infrastructure," he said. "Accurate fluid biomarkers, especially if they can be measured in blood, would be much more cost-effective and scalable."

Hansson and colleagues studied 682 individuals from the Swedish BioFINDER 2opens in a new tab or window cohort. The sample included 81 patients with Lewy body disease (Parkinson's disease or dementia with Lewy bodies), 40 with atypical Parkinsonian disorders, 214 with other neurodegenerative disorders like Alzheimer's disease, and 347 clinically unimpaired controls.

The researchers measured a panel of 2,943 proteins in CSF and 92 proteins in plasma using a highly sensitive, specific multiplex immunoassay. The primary goal was to identify biomarkers that could detect clinical Lewy body disease and atypical Parkinsonian disorders. The secondary aim was to find markers of preclinical Lewy body disease, defined as clinically unimpaired individuals with a positive alpha-synuclein seed amplification assay (SAA).

The researchers replicated their findings in an independent sample of 152 people to assess generalizability and measured candidate biomarkers in blood samples from 174 people.

Because the most common medication to treat Lewy body disease is levodopa combined with a DDC inhibitor (carbidopa), the researchers repeated their analyses in 45 people with early clinical disease who had not yet received dopaminergic drugs and found DDC levels were upregulated in this subgroup compared with controls (AUC 0.92).

DDC was not a good biomarker to discriminate atypical Parkinsonian disorders from Lewy body disease, Hansson and co-authors noted.

The alpha-synuclein SAA is an accurate biomarker for Lewy body disease but not always for atypical Parkinsonian disorders (which lack Lewy bodies), the researchers observed. "We suggest that DDC and alpha-synuclein SAA may be combined in clinical practice in the future, where high DDC and abnormal alpha-synuclein SAA would indicate Lewy body disease, whereas high DDC and normal alpha-synuclein SAA would indicate an atypical Parkinsonian disorder," they wrote.

A similar idea combining SAA with neurofilament light chain (NfL) has been proposed, Hansson and colleagues said. But unlike DDC, NfL is a "rather unspecific neurodegeneration biomarker that is affected across several neurodegenerative disorders, such as amyotrophic lateral sclerosis and frontotemporal dementia, among many others," they pointed out.

The study investigated only one technique to measure DDC protein levels in CSF or plasma, the researchers acknowledged.

"We need to develop laboratory tests that can be more easily used in research, and later on in clinical practice and trials," Hansson said. "Further, the results -- especially in blood -- need to be validated in further clinical relevant cohorts."

  • Judy George covers neurology and neuroscience news for MedPage Today, writing about brain aging, Alzheimer’s, dementia, MS, rare diseases, epilepsy, autism, headache, stroke, Parkinson’s, ALS, concussion, CTE, sleep, pain, and more. Follow

Disclosures

This work was supported by the Swedish Research Council, the Knut and Alice Wallenberg Foundation, Strategic Research Area MultiPark at Lund University, Swedish Alzheimer Foundation, Swedish Brain Foundation, Parkinson Foundation of Sweden, Cure Alzheimer's fund, Konung Gustaf V:s and Drottning Victorias Masonic Foundation, Skåne University Hospital Foundation, Regional Research Support, and the Swedish federal government.

Hansson reported relationships with ADx, AVID Radiopharmaceuticals, Biogen, Eli Lilly, Eisai, Fujirebio, GE Healthcare, Pfizer, Roche, AC Immune, Amylyx, ALZpath, BioArctic, Cerveau, Eisai, Genentech, Merck, Novartis, Novo Nordisk, Sanofi, and Siemens. A co-author declared relationships with ki elements/ADDF, Cytox, Eli Lilly, Roche, and Geras Solutions.

Primary Source

Nature Aging

Source Reference: opens in a new tab or windowPereira JB, et al "DOPA decarboxylase is an emerging biomarker for Parkinsonian disorders including preclinical Lewy body disease" Nat Aging 2023; DOI: 10.1038/s43587-023-00478-y.

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