Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Wednesday, September 20, 2023

Сause of Alzheimer's disease identified in new study

 Your doctor, if competent at all, should already known about ferroptosis from this research from September 2017.  And should have initiated stroke treatment interventions from it.

 Dementia research leads to potential new stroke treatment

The latest here:

Сause of Alzheimer's disease identified in new study

Researchers from the Oregon Health and Science University conducted a new study on the causes of Alzheimer's disease. They identified a unique form of cell death that operates like a snowball and rapidly impairs cognitive functions, according to the Annals of Neurology journal.During their study of post-mortem brain tissue from a dementia patient, scientists were able to identify a specific form of cell death in the white matter composed of myelinated axons, which accelerates cognitive decline.This particular type of cell death is known as ferroptosis and is induced by the accumulation and toxic influence of iron. In this process, immune brain cells in Alzheimer's and vascular dementia are destroyed. Immune brain cells called microglia die while performing their job of cleaning up "garbage" cells. However, if the "garbage" contains myelin particles rich in iron, it triggers a snowball effect, causing the death of other cells. Myelin creates a protective layer around nerves and gets damaged due to neurological stress. This leads to ferroptosis – the death of microglial cells in the brain's white matter.Borrow from Your Home, Not the Bank ($25k for $164/mo)"Everyone knows that microglia get activated to mediate inflammation. But nobody knew that they die in such large numbers. It's even strange that we didn't pay attention to this nuance before," write the authors of the study.>While suppressing ferroptosis is seen as a treatment goal for Alzheimer's, this new discovery may offer a completely different perspective on the development of new treatment methods for the disease. Perhaps scientists will be able to slow down the progression of neurodegenerative diseases. "We've missed the primary form of cell death in Alzheimer's and vascular dementia. We haven't paid enough attention to microglia as vulnerable cells and to the degradation of brain matter," emphasize the researchers. Now, pharmaceutical companies will have a new target - to develop compounds that limit microglial degeneration. This will pave the way for the creation of entirely new and effective drugs for the treatment of dementia and Alzheimer's disease.

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