Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Monday, December 5, 2011

Stroke risk factors

From Stroke July 1, 1997 vol. 28 no. 7 1507-1517
If they were a decent stroke association they would have the risk percentages and numbers of these occurring every year. If we don't have statistics/facts we can't figure out what is really a problem, similar to the color coded threat levels
http://stroke.ahajournals.org/content/28/7/1507.full
Table 1.Risk Factors for First Ischemic Stroke

Well-documented risk factors
Modifiable, value established
Hypertension
Cardiac disease
Atrial fibrillation
Infective endocarditis
Mitral stenosis
Recent large myocardial infarction
Cigarette smoking
Sickle cell disease
Transient ischemic attacks
Asymptomatic carotid stenosis
Potentially modifiable
Diabetes mellitus
Hyperhomocysteinemia
Left ventricular hypertrophy
Nonmodifiable
Age
Gender
Hereditary/familial factors
Race/ethnicity
Geographic location
Less well-documented risk factors
Potentially modifiable
Elevated blood cholesterol and lipids
Cardiac disease
Cardiomyopathy
Segmental wall motion abnormalities
Nonbacterial endocarditis
Mitral annular calcification
Mitral valve prolapse
Valve strands
Spontaneous echocardiographic contrast
Aortic stenosis
Patent foramen ovale
Atrial septal aneurysm
Use of oral contraceptives
Consumption of alcohol
Use of illicit drugs
Physical inactivity
Obesity
Elevated hematocrit
Dietary factors
Hyperinsulinemia and insulin resistance
Acute triggers (stress)
Migraine
Hypercoagulability and inflammation
Fibrin formation and fibrinolysis
Fibrinogen
Anticardiolipin antibodies
Genetic and acquired causes
Subclinical diseases
Intimal-medial thickness
Aortic atheroma
Ankle-brachial blood pressure ratio
Infarctlike lesions on MRI
Socioeconomic features
Nonmodifiable
Season and climate

Be careful out there

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