Deans' stroke musings: Drug found to reduce damage caused by heart attack and stroke

Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Saturday, June 8, 2013

Drug found to reduce damage caused by heart attack and stroke

But, but, have they accounted for the fact that mouse inflammation is not the same as human inflammation?
http://www.barchester.com/Media/News/2013/5/drug-found-to-reduce-damage-caused-by-heart-attack-and-stroke.htm
Research has found that a new drug can reduce the damage that heart attacks and strokes cause to the body.
Scientists at the Medical Research Council Mitochondrial Biology Unit inside Cambridge University found that a drug known as MitoSNO could repair tissue that has been starved of oxygen when running tests on mice.
After a heart attack, tissue can be damaged when blood flow is suddenly restored after a period without air.
Researchers believe MitoSNO has the power to turn off harmful molecules known as free radicals, which are produced during this process.
"MitoSNO effectively flicks a switch in the mitochondria, slowing down reactivation during those critical first minutes when blood flow returns and protecting the heart tissue from further damage," Dr Mike Murphy, who led the study, said.
He added that trials will need to be conducted on humans before anything conclusive can be stated, but if successful the drugs could be used in other areas of treatment.