Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Wednesday, May 10, 2017

Fish consumption, intake of fats and cognitive decline at middle and older age: The Doetinchem Cohort Study

I bet nothing here will ever make it into a diet protocol in your hospital or take home instructions post-stroke. Because your doctor and hospital don't ever read and translate research findings into usable protocols. Why would they? It would mean actually doing some difficult work summarizing the thousands of research articles on food. They would rather wait for SOMEONE ELSE TO SOLVE THE PROBLEM.  You are screwed once again.

Fish consumption, intake of fats and cognitive decline at middle and older age: The Doetinchem Cohort Study

European Journal of Nutrition
Nooyens ACJ, et al.
The Doetinchem cohort study was carried out to get insight into the effect of fish and fat consumption in the prevention of accelerated cognitive decline with aging, they tested relationship amongst fish and different fat consumption and 5–year change in cognitive functions. It was concluded that the higher cholesterol consumption was detrimental, while higher a-linolenic acid (ALA) consumption was beneficial for maintaining cognitive function with aging, already at middle age.

Methods

  • In 2612 men and women of the Doetinchem Cohort Study, aged 43–70 years at baseline, dietary consumption (including fish consumption) and cognitive function were evaluated at baseline and at 5–year follow–up.
  • In this study, average fish intake (frequency) and consumption (as energy percentages) of total fat, saturated, mono unsaturated, and polyunsaturated fatty acids (PUFA), linoleic, docosahexaenoic, eicosapentaenoic, and a–linolenic acid (ALA), and cholesterol were averaged over baseline and follow–up.
  • Consumption were studied in relation to 5–year change in global cognitive function, memory, information processing speed, and cognitive flexibility, utilizing ANCOVA and multivariate linear regression investigations.

Results

  • In this study, they observed no consistent relationship between (fatty) fish intake and cognitive decline.
  • They found higher cholesterol consumption was related to faster cognitive decline (p < 0.05).
  • Higher n–3 PUFA ( particularly ALA) consumption was related to slower decline in global cognitive function and memory (p < 0.01).
  • Consumption of other fatty acids were not related to cognitive decline
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