Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Sunday, December 6, 2020

Safety and efficacy of symptomatic carotid artery stenting performed in an emergency setting

I would think this whole problem could be eliminated by:

1. Determining if the Circle of Willis is complete.

2. If yes, then close the artery off to prevent ANY POSSIBILITY of clots breaking off and traveling to the brain.

3. This prevents stent placement complications per European Society of Cardiology

A - Minor complications
  • Carotid artery spasm
  • Sustained hypotension / bradycardia
  • Carotid artery dissection
  • Contrast encephalopathy (very rare)
  • Minor embolic neurological events (TIAs)
B - Major complications
  • Major embolic stroke
  • Intracranial hemorrhage
  • Hyperperfusion syndrome
  • Carotid perforation (very rare)
  • Acute stent thrombosis (very rare)
  • Complications at the site of the vascular access

Don't listen to me, I'm not medically trained and I don't have a Dr. in front of my name.  But pepper your doctor with lots of questions, including GUARANTEEING NO PROBLEMS. Why would you want to place an inflexible stent in a flexible artery? Hell, my right carotid artery was 80% blocked at time of stroke which my doctors never found so luckily they didn't try to stent me. It eventually completely closed on its own with absolutely no cognitive problems encountered.

Don't listen to what I have to say, I'm not medically trained.

The latest here:

Safety and efficacy of symptomatic carotid artery stenting performed in an emergency setting

First Published December 6, 2020 Research Article 

Carotid artery stenting (CAS) has increasingly emerged as an alternative strategy to carotid endarterectomy in the treatment of patients with symptomatic carotid stenosis. Optimal timing for CAS after symptoms onset remains unclear. We aimed to evaluate the safety and efficacy of CAS when performed in an emergency setting.

We performed a retrospective analysis of CAS patients admitted to our CSC with symptomatic extracranial carotid occlusion or significant stenosis from January 2014-September 2019. Emergency CAS was defined as CAS performed during the same hospitalization from TIA/stroke onset, whereas elective CAS as CAS performed on a subsequent admission. The primary outcome was defined as the occurrence of any stroke, myocardial infarction, or death related to the procedure at 3 months of follow-up. Secondary outcomes included periprocedural complications and the rate of restenosis/occlusion at follow-up. Logistic regression and survival analyses were used to compare outcomes and restenosis at follow-up.

We identified 75 emergency and 104 elective CAS patients. Emergency CAS patients had significantly higher rates of ipsilateral carotid occlusion (17% vs. 2%, p < 0.001) and use of general anesthesia (19% vs. 4%, p = 0.001) than elective CAS. There were no significant differences between emergency and elective CAS in the primary (5.7% vs. 1%, p = 0.161) and secondary (9% vs. 4.8%, p = 0.232) outcomes. We did not find differences in the rate of restenosis/occlusion (7% vs. 11.6%; log-rank test p = 0.3) at a median of 13 months follow-up.

In our study, emergency CAS in symptomatic patients might have a similar safety and efficacy profile to elective CAS at 3 months and long-term follow-up.

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