Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Tuesday, February 16, 2021

Takotsubo cardiomyopathy in acute ischemic stroke

 Something else for you to worry about, your doctor having knowledge of this.

Diagnosis of Takotsubo Cardiomyopathy– Mayo Clinic Criteria –

The latest here:

Takotsubo cardiomyopathy in acute ischemic stroke

First published: 07 August 2008
Citations: 126

Abstract

Objective

Takotsubo cardiomyopathy, which is characterized by transient left ventricular apical ballooning, is a known complication of subarachnoid hemorrhage. The aim of this study was to identify the clinical characteristics of acute ischemic stroke patients who experienced development of takotsubo cardiomyopathy.

Methods

Seven patients who were diagnosed as having takotsubo cardiomyopathy based on their electrocardiographic and echocardiographic findings were studied. They were selected from among 569 consecutive patients who were admitted to our stroke center within 24 hours after onset of acute ischemic stroke. The findings of nine previously published cases were also reviewed.

Results

All seven patients were women, and six were 75 years or older. The initial National Institutes of Health Stroke Scale score ranged from 3 to 28. The culprit infarcts included or were close to the insular cortex in six patients and were located extensively in the vertebrobasilar arterial territory in the other patient. Abnormal findings on electrocardiographic monitoring appeared within 10 hours after stroke onset in five patients and at 6 and 12 days, respectively, in the other two patients. The cardiomyopathy was symptomatic in only two patients. Plasma brain natriuretic peptide levels exceeded the upper normal limit by 10‐fold in all patients. The previously published cases were mostly women and had mainly vertebrobasilar stroke.

Interpretation

Takotsubo cardiomyopathy is not a rare complication of acute ischemic stroke. It most often occurred soon after stroke onset and was commonly asymptomatic. Female sex and insular damage were predominant features of the stroke patients who experienced development of takotsubo cardiomyopathy. Ann Neurol 2008

 

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