Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Sunday, July 24, 2022

Long-term Changes in Depressive Symptoms Before and After Stroke

Hopefully you have enough working brain cells to recognize that 100% recovery protocols would prevent post stroke depression. Looking forward to your research on that, since this depression research will not be needed.

Long-term Changes in Depressive Symptoms Before and After Stroke

Maria Blöchl, Steffen Nestler

 

Abstract

Objectives: To determine the trajectory of depressive symptoms several years before and after incident stroke.

Method: We analysed data from 10,797 participants from the English Longitudinal Study of Ageing (ELSA) without a history of stroke at baseline (wave 1). We matched participants with incident stroke during the 12-year follow-up (waves 2-7) to stroke-free individuals using propensity scores accounting for age, gender, education, ethnicity, and vascular risk factors. Trajectories of depressive symptoms before and after stroke were analysed using multilevel models.

Results: Among the 10,797 participants (mean age 64.6 ± 9.9 years, 54.8 % women), we identified 425 individuals with incident stroke. At the assessment before stroke, these individuals demonstrated an increase in depressive symptoms compared to matched controls. There was a further increase in depressive symptoms in stroke survivors after the acute event, which persisted for several years. Symptom-level analyses revealed that differences in depressive symptoms between stroke survivors and stroke-free controls before and after stroke were most pronounced for mood- and fatigue-related symptoms.

Discussion: Incident stroke is associated with long-term increases in depressive symptoms. A small part of this increase occurs in the years before stroke, perhaps indicating the incipient pathological process. Particular attention should be paid to depressive symptoms in the long-term care of patients, and especially to fatigue-related symptoms.

  • Received June 11, 2021.
  • Accepted in final form April 5, 2022.

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