Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Thursday, July 7, 2022

Angiotensin receptor blockers may lower risk for progression to dementia

I had to look up mine separately; nifediprine, is in a class of medications called calcium-channel blockers

 
Examples of angiotensin II receptor blockers include:
  • Azilsartan (Edarbi)
  • Candesartan (Atacand)
  • Eprosartan.
  • Irbesartan (Avapro)
  • Losartan (Cozaar)
  • Olmesartan (Benicar)
  • Telmisartan (Micardis)
  • Valsartan (Diovan)

 

Angiotensin receptor blockers may lower risk for progression to dementia

Angiotensin receptor blockers may reduce progression from mild cognitive impairment to dementia in patients undergoing antihypertensive treatment, researchers wrote in Hypertension.

Compared with ACE inhibitors, beta-blockers, calcium channel blockers, diuretic and no treatment at all, antihypertensive treatment using angiotensin receptor blockers (ARBs) was associated with lower risk for progression to dementia during a median of 3 years follow-up among patients with mild cognitive impairment and hypertension, according to the researchers.

Man trying to think
Source: Adobe Stock

“The angiotensin hypothesis has recently been proposed that the renin-angiotensin system plays a role in brain function. ... Medications that increase angiotensin-mediated activity at the angiotensin II and angiotensin IV receptors (eg, ARBs) may provide better brain protection compared with those decreasing activity at these receptors (eg, ACE inhibitors),” Zhenhong Deng, of the department of neurology at Sun Yat-sen Memorial Hospital, Sun Yat-sen University in Guangzhou, China, and colleagues wrote. “It remains unclear whether and to what extent ARBs are superior to ACE inhibitors in reducing progression to dementia in patients with mild cognitive impairment.”

Researchers investigated whether ARBs, compared with ACE inhibitors and other antihypertensive medications, may lower risk for progression from mild cognitive impairment to dementia in patients with hypertension.

Researchers used the Alzheimer’s Disease Neuroimaging Initiative, developed by the Laboratory of Neuro Imaging at University of Southern California, to identify 403 patients with hypertension and mild cognitive impairment at baseline (mean age, 74 years; 38% women). Data on antihypertensive medications received during a median follow-up of 3 years were self-reported.

During follow-up, 39.2% of participants progressed to dementia and the 3-year rate of progression-free survival was 67%.

In patients with hypertension and mild cognitive impairment, ARBs were associated with lower risk for progression to dementia compared with ACE inhibitors (adjusted HR = 0.45; 95% CI, 0.25-0.81; P = .023).

Compared with beta-blockers, calcium channel blockers and diuretics, ARB use was associated with lower risk for progression of mild cognitive impairment to dementia (aHR = 0.49; 95% CI, 0.27-0.89; P = .037).

Hypertension treatment with ARBs was also associated with lower risk for progression to dementia in patients with hypertension and mild cognitive impairment compared with no treatment (aHR = 0.31; 95% CI, 0.16-0.58; P = .001).

In adjusted analyses, treatment with ACE inhibitors did not affect progression of cognitive impairment compared with beta-blockers, calcium channel blockers and diuretics (P = .685) nor compared with no treatment (P = .179).

Moreover, antihypertensive treatment with beta-blockers, calcium channel blockers and diuretics did not prevent cognitive decline compared with no treatment in this cohort (P = .121).

“Overall, angiotensin II and IV may provide neuroprotection through angiotensin II and angiotensin IV receptors, although most evidence was based on experimental studies and the mechanisms were not fully understood,” the researchers wrote. “ARBs, which selectively block the angiotensin I receptors without inhibiting ACE and result in the relatively upregulated activities of angiotensin II and angiotensin IV receptors, and keep the pathway of amyloid beta degradation mediated by ACE intact, may offer superior protection than simultaneously lowering all the angiotensin receptors’ activities with ACE inhibitors.”

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