Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Tuesday, July 17, 2018

Is spasticity or spastic cocontraction of the elbow flexors associated with the limitation of voluntary elbow extension in adults with acquired hemiparesis?

I don't care that spasticity causes the problem of elbow extension. What the fuck is the intervention solution? Don't researchers understand survivors want solutions NOT descriptions of our problems?
Where the fuck is the strategy leading to solutions?
https://www.sciencedirect.com/science/article/pii/S1877065718310947

Introduction/Background

Muscle overactivity, including spasticity and spastic cocontraction, is an involuntary motor unit recruitment participating in the spastic paresis syndrome after cerebral injury. Spasticity is defined as velocity-dependent increase in tonic stretch reflexes. Spastic cocontraction refers to increased antagonist muscles recruitment triggered by the volitional command of agonist muscles. This study aimed to clarify the association between spasticity and spastic cocontraction of elbow flexors and to study their contribution to the limitation of active elbow extension in hemiparetic adults.

Material and method

Ten adults with acquired hemiparesis and ten healthy participants were included. Surface EMG recorded from elbow muscles during elbow isometric extension contractions was used to compute the index of cocontraction (ICC) for each participant, while spasticity, limitation of active elbow extension, and upper extremity Fugl-Meyer Assessment (FMA-UE) score were obtained in hemiparetic participants. Non-parametric Spearman correlations were performed to investigate the relationship between ICC and (i) limitation of active elbow extension, (ii) elbow flexors spasticity and (iii) FMA-UE.

Results

Our results showed significant ICC in three hemiparetic participants compared with healthy participants, and significant associations between cocontraction and (i) active elbow extension limitation (rs = 0.81, P < 0.001) and iii) Fugl-Meyer Assessment score (rs = −0.53, P = 0.017) in hemiparetic participants. No significant correlation was found between spasticity and active elbow extension limitation.

Conclusion

Our results are the first to show that spastic cocontraction directly contributes to elbow extension deficit in adults with acquired hemiparesis, and further confirm that spasticity and spastic cocontraction have different functional repercussions with regards to impaired motor function. Our findings support the conclusion that spastic cocontraction, rather than spasticity, has significant functional repercussions on impaired active motor function in hemiparetic adults. Therapeutic innovations should be directed toward reduction of spastic cocontraction to improve motor function in acquired hemiparesis.

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