Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Friday, April 20, 2012

Mild Plaque May Pose Big Risk

So if your stroke was Cryptogenic(unknown origin) you might want to consult your doctor again.
 http://www.medpagetoday.com/Cardiology/Strokes/32190

Are Cryptogenic Strokes Really Cryptogenic?
More than one-third of patients who had a stroke of unknown origin were found to have complex carotid plaques in nonstenosed arteries, according to a small study.
MR imaging showed the prevalence of complicated type VI plaques, according to American Heart Association classification, in 12 of 32 carotid arteries ipsilateral to the stroke, compared with no such findings in the contralateral arteries, reported Tobias M. Freilinger, MD, from Ludwig-Maximilians-University Munich, and colleagues.
In three-quarters of the plaques, intraplaque hemorrhage was the most common diagnostic feature, followed by fibrous plaque rupture in 50%, and surface thrombus in 33%, according to results published in this month's JACC: Cardiovascular Imaging.
"The realization that even apparently 'minor' atherosclerosis may harbor high-risk disease should change our perception of what constitutes risk for a patient," wrote Alan Moody, MD, from Sunnybrook Health Sciences Center in Ontario, Canada, in an accompanying editorial.
Also reported in the journal, Alistair C. Lindsay, MBChB, from the University of Oxford in the U.K., and colleagues found that slightly more than half of 41 patients who suffered a transient ischemic attack had type VI plaques compared with eight asymptomatic controls.
Lindsay's group identified the three most common features as intraplaque hemorrhage, cap rupture, and surface thrombus. These features were in carotid arteries that were not considered significantly stenotic.
They also found that only two plaques showed signs of healing at 6-weeks' follow-up. Moody said that "this represents an ongoing source for thromboemboli and a potential therapeutic target for secondary prevention."

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