Excuses, excuses on understanding spasticity.
http://www.hubmed.org/display.cgi?uids=23319482&utm_source=feedburner&utm_medium=feed&utm_campaign=Feed%3A+nih%2Fbxxu+%28Stroke+rehabilitation%29&utm_content=Google+Feedfetcher
Spasticity is defined clinically by increased muscle tone and tendon
jerk hyperreflexia in patients who are at rest. However, the
excitability of spinal circuits changes during movement, and this
definition provides no insight into the extent to which spasticity and
associated motor disturbances cause disability. Only a few spinal
circuits have been shown to underlie the abnormalities of patients at
rest. Movement can be restrained by pathologically enhanced muscle tone,
and there is defective control of the feedback to active motoneurons
through virtually all spinal reflex pathways. Spasticity does not
necessarily require treatment:(Bullshit, how many survivors have you consulted?) in fact, some patients rely on the
increased muscle tone to help support otherwise weak muscle contractions
for stance and locomotion. In addition, much of the increase in muscle
tone arises from changes in muscle and motor units, independent of
reflex mechanisms. Managing a patient with impairment after a stroke
requires therapy tailored to that particular patient because the
mechanisms contributing to the disability experienced by one patient may
differ from those affecting another.(Damn you're good at giving excuses for not being able to help survivors. Will you give the same answer to your mother?)
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